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  • in reply to: The Vaxx is Dead. Now What? #89530

    My wife knows I’m deep in the weeds in trying to sort this stuff out and she dropped me a video to check out.

    wife: “Have you checked it”

    me: “Not yet.”

    wife: “You really need to. It has immune blood panel info for before, after jab 1 and after jab 2.”

    That was Wednesday night.

    response last night got me curious about poison ivy and topical IVM. Which led to an article on the immune system and the impact of toxins. To which I noted the following:

    T-cells participate directly as killer cells against virally infected host cells.

    T-cells are long-lived lymphocytes. Some estimates of longevity place some T-cells as existing for 15-20 years.

    Since there is little replacement of T-cells, alterations in this compartment may be very detrimental. Lesions in the T-cell system result in enhanced susceptibility to viral, fungal and para-sitic infections and alter the levels of most anti-body responses.

    Then @expatkiwi posts a link to Alex Berenson latest. The slope change on that curve is flashing red.

    Raul, pens “The Vaxx is Dead, Now What?” Another perfect summary and snapshot of where this train wreck is at/headed.

    I finally check out that video my wife sent. The real time stream of consciousness in this community never ceases to amaze me.

    in reply to: Debt Rattle October 7 2021 #89465


    That’s neat. Good to know. Thanks.

    in reply to: Debt Rattle October 7 2021 #89447

    thanks, wdt, scrapper,

    Let’s try it this way, can a human apply it topically and achieve a therapeutic benefit?

    in reply to: Debt Rattle October 7 2021 #89436

    IVM applied topically on the skin, has this been discussed?
    I got an email from a coworker about this. Is this alternative way to get it in the system?

    in reply to: Debt Rattle October 7 2021 #89432

    Yellow buttons. I guess it’s been nearly 90 years but I did not think it was so easily forgotten. What’s happening in Germany and Israel is surreal, let alone Australia.

    Back when the local stores were requiring masks and had checkers at the entry, my wife went to a Trader Joe’s in the area. There was a line since they were limiting the number of shoppers in the store. She was maskless and when she got to the entry, they told here to mask up.

    She proceeded to school them on the law (as there was no enforceable law where we live). They then told her she would have to push around a cart with a tall flag attached to it. She asked “What is that for?” They said it was so the other shoppers could see her coming over the aisles. She was like, “and yellow stars sewn on my clothes are next?”. They seemed confused, to which she said “Hello, Nazi Germany, concentration camps, WW2.” Not only did she get to shop without mask or a flag, many other shoppers thanked her for standing her ground.

    There are only two wills active in the world. Most are unconscious to the one that will kill their soul. Today, I am getting that conscious Jew vibe int the 1930s, today I weep.

    in reply to: Attack on Red Blood Cells #89352

    John, that was Raul’s genius. I have learned much following the daily links, articles and the commentary provided by you and others.

    in reply to: Attack on Red Blood Cells #89351


    Crazy as it may seem I do this when I get sick. I stumbled across this many years ago and it works well, but seriously make sure you don’t get blow back in the ear tubes if you attempt this. It’s called the flip turn flush. I have only been on oral antibiotics twice in my life (penicillin for kidney infection in grade school and zpak for sinus infection 20 years ago) Not sure how I would incorporate IVM.

    Also, following up with Xlear several times a day. This stuff is amazing in my experience.

    Xlear | Sinus Care

    in reply to: Attack on Red Blood Cells #89343


    She is not the real deal which is obvious from her website (I commented to michael reid on this last night). I am ok with her being my nurse but definitely not my doctor. When blood is drawn in a test tube, there is always some liquid chemical in the bottom so that it doesn’t clot on way to the lab. I doubt she even knows that. Anyway here is a proper video on what clotting blood looks like.

    Raul, thanks for putting that together. Much appreciated and humbled.

    in reply to: Debt Rattle October 5 2021 #89301


    Most anybody can use a microscope.

    Her qualifications are not even recognized in the her home country of South Africa.

    Just a hunch, but I don’t think she is being honest.

    in reply to: Debt Rattle October 5 2021 #89290

    Yoh Michael,

    I would think Stew would do a better job to prevent from getting punked. “How many surgeries did say she performed in a day in that interview” From her website.

    “Please take note that Dr Botha is not a medical Dr but an academic Dr. Her qualification is recognized in 194 countries including America and Europe but not in SA. Dr Botha does not diagnose or treat any illness but supports health through complementary medicine, lifestyle management.”

    in reply to: Debt Rattle October 5 2021 #89269

    Germ, triathletes have efficient cardiovascular systems. The velocity of the blood in the capillaries next to the air sacs is low to begin with and in an athlete even lower. The fact that those cells happened to to get painted for destruction by the latent immune response is no surprise to me in the way I have been thinking thru this lately. I think I will keep running with it (pun intended).

    in reply to: Debt Rattle October 5 2021 #89268

    zerosum, yep there are others that will see this as raw meat and unpack. I threw it out there in hopes of that. In, the moment, I am trying to piece together other stuff.

    in reply to: Debt Rattle October 5 2021 #89257

    Phoenix vice: (the auto complete did that) kinda funny no offense intended. Absolutely, the injections introduce a bit of glycoprotein manufacturing apparatus in to the blood stream. It has a head start over the rest that either stays in the muscle or drains into a nearby lymph node ultimately a fox and hound scenario ensues. The area associated with with all the endothelial cells that make up our vascular system is on the order of a football field. So the damage could occur anywhere. Inflammation, stroke, clots, hamster nuts. Then there is the CNS and it looks Like there is no place that theses chards of glycoproteins can go, Blood brain barrier be damned. Then there is fundamental immune cell-cell communication associated with glycoproteins that in the case of cancer is may also be disrupted.

    in reply to: Debt Rattle October 5 2021 #89254

    Mr. House, I get the whole numerator/denominator, jigging relationships to look like hockey sticks instead of plateaus and yes they are lying to build back better or great reset. Or maybe the CCP virus plus uploaded CCP mRNA sequence is a set up for MERS-Cov 2.0. It’s just a thought experiment into unintended or intended consequences and how we may be blind to the outcome. As @Antidote rightly reminds us, “we only see what we look at”

    in reply to: Debt Rattle October 5 2021 #89250

    Here is a presentation of a study that focuses on Medicare recipients 65 and over.

    My thoughts on Chart 16

    In the trials that established the baseline efficacy, people 65 and over weren’t included

    Wasn’t there a lawyer that obtained death numbers in the Medicare system of people that died before they were considered fully jabbed up. How would that change this chart if we considered these as breakthroughs infections that were hospitalized?

    And Chart 17

    Impressive risk reduction for those who previously had Covid and got the jab. Likely, has more to do with natural immunity. This group of people are likely skewing the benefit on the previous chart.

    But here is the thing, what if ADE is not a thing and nobody starts melting in their footprint like WTC7. In the limit, as more and more unvaccinated people that have had Covid get jabbed will the efficacy numbers start to inflect making boosters look like they are improving things. I know, Its really natural immunity that’s the driver but the data will be used to give credit to the jab.

    in reply to: Debt Rattle October 3 2021 #89129

    Veracious Poet, 38 years ago I was 18 and on track to become a good corporate soldier. No wonder it feels like the Constitution and Bill of Rights are null and void. Your commentary is always appreciated even though the mood elevator can take a hit.

    On our way out for a ride tonight, my wife was talking about not having carpets when we move. I said, “Babe, we might have to get used to dirt floors.”

    But ya know, I feel a new kind of freedom. Made some new friends with some amazing people that under the old normal I would have never met. My wife’s mom lived with us her last year of life. If it wasn’t for Covid and the locking down of senior care facilities we would have never got that opportunity. Our ride ended with a double rainbow. How many of those do you see in a lifetime.

    I am sure Louis experienced the some of the worst aspects of what his fellow man was capable of but yet it brought the best in him for all to enjoy.

    in reply to: Debt Rattle October 3 2021 #89105

    We got our “get the jab email” on Friday.

    In this email they said that we will follow the law. Sorry, this is not a law. It is an Executive Order from the President. It only covers federal employees under the direct control of the Executive Branch (DHS, DOD, DOJ, etc.). It only applies to those contractors that work directly for the federal government in the Executive Branch controlled agencies. It does NOT apply to privately owned defense contractor companies that have been awarded defense contracts. So our company lawyers are playing it a little loose with wording to force the mandate. That being said, if we ever want to win another US defense contract, the company will do what ever the government recommends, regardless of its ethical or legal standing.

    They set the date for Dec. 8th. I don’t think that date is unique to us but was set by the Biden administration. Hey, one day after the Pearl Harbor day. They really want to liken this to a war.

    Non-negotiable, never felt so alive.

    in reply to: Debt Rattle October 3 2021 #89104

    Raul, email sent. Thanks

    in reply to: Debt Rattle October 3 2021 #89087

    @Raul “can you turn your research into an article? I can help with the writing if needed”

    I would need your help, you have an amazing gift/talent for doing that.

    in reply to: Debt Rattle October 2 2021 #89055


    Will do, this infection can really set you back making the recovery frustrating. In article [1], the long term impact on WBCs was a new one for me. I haven’t dug too deep here yet. My wife is nearly recovered, still seems to have a bit of walking pneumonia and fatigue. Just got some blood work back and her thyroid was impacted. Big swing towards the hyper end of the range.

    The human body is a universe in and of itself and each aspect of it is a galaxy.

    in reply to: Debt Rattle October 2 2021 #89052

    In regards to IVM studies.

    1) There may be little impact in a respiratory only infection, therefore taking it prophylactically may not help much in the respiratory phase if infected.

    2) The real impact seems to be for those whose infections have moved into the vascular system.

    in reply to: Debt Rattle October 2 2021 #89051

    Anyway, before addressing the best theory I have come across regarding Covid in the blood I wanted to circle back and highlight the spike proteins RBD affinity to Sialic Acid (SA) and its importance in the human body. Here is the image, showing how this affinity grows from a Corona cold virus thru Sars-Cov2.


    Below is from the abstract of the linked paper. (It’s mainly for reference if interested)

    Sialic acid play important roles in human physiology of cell-cell interaction, communication, cell-cell signaling, carbohydrate-protein interactions, cellular aggregation, development processes, immune reactions, reproduction, and in neurobiology and human diseases in enabling the infection process by bacteria and virus, tumor growth and metastasis, microbiome biology, and pathology. It enables molecular mimicry in pathogens that allows them to escape host immune responses.

    Viral sialic acid-recognizing lectins or HAs can agglutinate RBC. Viruses use sialic acids linked to glycoproteins and gangliosides to attach to host cells, followed by their entry, for example, corona virus, DNA tumor viruses, hepatitis virus, influenza viruses (A, B, and C), mouse polyoma virus, mumps, Newcastle disease virus (NDV), norovirus, parainfluenza viruses, rotavirus, and Sendai virus. HAs from influenza A, C, NDV, and polyoma viruses have been crystallized. Sialic acid-recognizing lectins from adenoviruses and picornaviruses have not been identified.

    Some of these viruses carry neuraminidase or sialyl-O-acetyl-esterase that destroys the receptor, promotes virus release from infected cells, and removes sialic acid on host cell affecting cell surface binding of the virus. Influenza A virus enters the host by using host surface sialic acids. Influenza C virus HA-esterase specific for 9-O-acetylated sialic acids can break down 9-O-acetyl ester. HA-esterase from mouse hepatitis virus is specific to sialic acids substituted by O-acetyl group at the C-4 position (Neu4,5Ac2). HA-neuraminidase of NDV84 and parainfluenza viruses perform vital functions in infection biology

    Back in Oct2020, independent researcher David Scheim, uploaded this paper to SSRN. It is not peer reviewed. In virus time it was before the rollout of the jabs and early days of mass IVM awareness. Here is the link to the abstract. From there you can open the pdf.


    The role of vascular occlusion in the morbidities, pulmonary and systemic, of COVID-19 has received increasing focus. Histological studies of lung tissue from COVID-19 patients have found extensively damaged endothelium of capillaries adjoining relatively intact alveoli, corresponding to hypoxemia accompanying normal breathing mechanics in such patients. Advanced image analysis of lung CT scans of COVID-19 patients reveals redistribution of blood flow from smaller to larger diameter blood vessels, this effect correlated with the degree of breathing dysfunction.

    Essential to the study of vascular occlusion in COVID-19 are viral properties dating back to studies of Jonas Salk in the 1940s that have been positively established for SARS-CoV-2.

    First, SARS-CoV-2 binds to red blood cells (RBCs), in vitro and also clinically in COVID-19 patients.

    Second, although fusion and replication of SARS-CoV-2 occur via ACE2, such hemagglutinating viruses initially attach to infective targets and clump with blood cells via much more abundantly distributed sialic acid (SA) glycoconjugate binding sites. SARS-CoV-2, in particular, attaches to these SA sites.

    Third, certain enveloped viruses express an enzyme, hemagglutinin esterase (HE), that counteracts viral-RBC clumping. Notably, among betacoronaviruses, the common cold strains express HE while SARS-CoV-2, SARS-CoV-1 and MERS, the virulent strains, do not.

    These hemagglutinating properties of SARS-COV-2 establish a framework for “catch and clump” induction of microvascular occlusion proposed here. Ultramicroscopic studies of tissues from COVID-19 patients indicate a key role for hemagglutination early and mid-course in COVID-19, before such clumps harden into clots via the coagulation cascade. Hemagglutination may be reversed by two anti-COVID-19 therapeutics that each competitively bind to SARS-CoV-2 spike protein, blocking such viral attachments. One therapeutic is antiviral antibodies generated by vaccines, the anti-hemagglutination effect of which is exhibited in Jonas Salk’s hemagglutination inhibition assay. The other therapeutic is ivermectin (IVM), a drug of Nobel Prize honored distinction, distributed in 3.7 billion doses worldwide. In ten clinical trials, three with randomized controls, IVM yielded mortality reductions for COVID-19 of 90% at highest doses. IVM may limit virulence of SARS-CoV-2 by steric interference with multivalent spike protein attachments to SA binding sites, blocking hemagglutination, an effect likely to target mutant viral strains.

    in reply to: Debt Rattle October 2 2021 #89050

    I had to switch from “be the spike” to “be the Lipid Nano Particle” and I now find myself in a vortex of understanding for which there may be no escape velocity. It could be a kool-aid vortex, but we will see. Suffice it to say, though the primary mechanisms are different, the understanding gained on the infection side is still most useful.

    The inflection gains access to the blood stream (“ARDS” like DAD not a precursor)
    1) The virus gains access to an epithelial cell in the air sac of the lungs by attaching to ACE2. It uses this cell to make many copies of itself.
    2) Once it breaches the epithelial wall, which is only one cell thick it gains access to an ACE2 receptor on the pericytes, which sit on top of the endothelial cells that make up the nearby capillaries.
    3) Many more copies are made, weaking the capillary walls, which again are only one cell thick. The virus now has access to the blood stream.

    Here are three interesting articles, starting with the most recent, describing complications related to having a spike coated virus in the blood.

    [1] (virus time = Jul2021)

    “Shortness of breath, fatigue and headaches, some patients still struggle with these long-term effects of a severe infection by the SARS-CoV-2 coronavirus after six months or more. This post COVID-19 syndrome, also called COVID long-haulers, is still not properly understood. What is clear is that during the course of the disease, often blood circulation is impaired, clotting and dangerous vascular occlusions can occur, and oxygen transport in is limited. These are all phenomena in which the blood cells and their physical properties play a key role.”

    “They found that, for example, the size and deformability of the red blood cells of patients with the disease deviated strongly from those of healthy people. This indicates damage to these cells and could explain the increased risk of vascular occlusion and embolisms in the lungs. In addition, the oxygen supply, which is one of the main tasks of the erythrocytes, may be impaired in infected persons.”

    Lymphocytes (one type of white blood cell responsible for the acquired immune defense) were in turn significantly softer in COVID-19 patients, which typically indicates a strong immune reaction. The researchers made similar observations for neutrophil granulocytes, another group of white blood cells involved in the innate immune response. These blood cells even remained drastically altered seven months after the acute infection.

    [2] (virus time = Feb2021)

    COVID-19 can affect the blood. Its spike protein may be the culprit.

    In this article they refer to RGD, I think they mean RBD (Receptor Binding Domain)

    The autopsy reports revealed COVID-19 patients were suffering from huge amounts of thick, coagulated blood, and dysfunctional blood vessels were tearing through body tissue instead of repairing it—highly uncommon side effects of respiratory diseases.

    The postmortem evidence plus his own experience with something called “COVID toes”—an odd side effect of the disease that causes heightened blood vessel formation in the toes, turning them bright red—led Makowski to speculate that something about the virus might be causing abnormal blood-related complications.

    “One of the most perplexing and devastating effects of this disease is the scenario where three or four weeks after being hospitalized with pneumonia, people under the age of 50 are back home, they feel fine, and then all of a sudden they have a stroke and die,” says Makowski, professor and chair of the bioengineering department at Northeastern.

    [3] (virus time = Nov2020)

    The oxygen-saturation level of COVID patients, especially those with severe cases, was prone to dropping to dangerous levels, even below 90%. “We asked the question: Can it be due to the cell that transports oxygen?” D’Alessandro said. “Can COVID attack red blood cells – the most abundant cell in the human body – which has evolved specifically to transport oxygen?”

    The answer to both questions was “yes.” The study revealed that SARS-CoV-2 damages the membranes of oxygen-carrying red blood cells. The virus didn’t affect the cells’ hemoglobin, which allow the cells to pick up oxygen, but it did damage membrane proteins responsible for blood cell structure, a characteristic that allows these cells to indirectly regulate red cell capacity to release oxygen and, most importantly, to squeeze through narrow capillaries in the periphery of the bloodstream.

    “When the red blood cells are damaged, and you’re a COVID patient who is exposed to another stress – pharmacological treatment, high fever or, after recovering from the disease, exercise or something of that nature – then your red blood cell is more likely to hemolyze (rupture),” D’Alessandro said.
    This explains why D’Alessandro, who jumped back into training for a late-summer marathon, noticed that more than a month after his initial COVID bout – two subsequent tests came out negative – he felt extremely tired just 20 minutes into a run.
    “Something was happening in the red blood cells, and it’s why we believe that they are part of the problem in long-term COVID symptoms,” he said.

    The lingering oxygen-level difficulties are explained by the lifespan of red blood cells. The cells circulate for up to 120 days before the body replaces them. To make room for hemoglobin, red blood cells have evolved to lose nuclei and organelles that allow other cells to replace damaged molecular components. So, if the virus damages red blood cells, it will be up to four months before they are cleared and replaced with cells that do not carry such damage.

    in reply to: Debt Rattle October 2 2021 #89006

    Dr. D = hot knife thru butter.l

    A truly wise person uses few words; a person with understanding is even-tempered.

    in reply to: Debt Rattle October 2 2021 #89003

    Question. In this Japanese government report on the bio distribution of lipid nano particles in rats, how should I interpret the blood related levels (bottom of chart) and someone’s decision not to report the % of Administered dose for the blood related levels (bottom right)?

    Given that whole blood contains the liquid fraction of blood (i.e., plasma) as well as the cellular elements that lead to clotting under certain circumstances. Which include red blood cells (RBCs), white blood cells, and other components.

    Should I smell a rat?


    in reply to: Debt Rattle September 29 2021 #88812

    Kind of looking forward to the future. The last time oligarchical control was replaced following a pandemic with an emphasis on freedom of the human spirit was the Black Death.

    in reply to: Debt Rattle September 29 2021 #88807

    Doc Robinson: “Intravenous injection of COVID-19 mRNA vaccine can induce acute myopericarditis in mouse model“, then what is a more likely (and better supported) cause of the damage?”

    I think I will go for the “more likely” in the human model as we are meant to be the true guinea pigs. I have a few gaps in my theory. I either need to be shot down and put out of this quantum entanglement obsession I have with TAE or a bit of help filling in the final gaps.

    Keep in mind my wheel house is aero/thermodynamics and nothing to do with this. I have been involved in more than my fair share of root/probable cause analyses so I am drawn to puzzles no matter what the subject matter. I think I have a grip on how to cast this. First I will state my theory and then back fill it with the supporting information as I get time to do so.

    Basically this is the core of what I believe to be happening. Once the cells with surface spike glycoproteins enter the bloodstream, whether infection or injection, they will bind to the red and white blood cells. They will go thru the process of attaching and detaching depending on where they are in the vascular system. Clumps or clots can build up anywhere, precipitating any number of undesirable outcomes. The immune system does not particularly see them as invaders and they will eventually cycle out of our system as new cells are made to replace them. The measured immune response from the jab has little to do with preventing infection and also wanes as the life cycle of the impacted red and white blood cells is completed. The injections are worthless and they down regulate the immune system while the cells they produce are circulating.

    in reply to: Debt Rattle September 29 2021 #88795

    Loved the responses from the NBA players to the press, especially Bradley Beal. I faintly heard the reporters amygdala short circuiting.

    in reply to: Debt Rattle September 28 2021 #88723

    Biden to fine companies $70,000 To $700,000 for violations of Vaccine Mandate…
    Posted by Kane on September 28, 2021 8:21 pm

    Biden’s Vax Mandate To Be Enforced By Fining Companies $70,000 To $700,000

    On Saturday, Speaker Nancy Pelosi’s House quietly tucked an enforcement mechanism into their $3.5 trillion “reconciliation” bill, passed it out of the Budget Committee, and sent it to the House floor.

    Buried on page 168 of the House Democrats’ 2,465-page mega bill is a tenfold increase in fines for employers that “willfully,” “repeatedly,” or even seriously violate a section of labor law that deals with hazards, death, or serious physical harm to their employees.

    The increased fines on employers could run as high as $70,000 for serious infractions, and $700,000 for willful or repeated violations—almost three-quarters of a million dollars for each fine.

    If enacted into law, vax enforcement could bankrupt non-compliant companies even more quickly than the $14,000 OSHA fine anticipated under Biden’s announced mandate.

    in reply to: Debt Rattle September 28 2021 #88722

    Looks like I didn’t link to #4 correctly, here is Bertozzi.

    in reply to: Debt Rattle September 28 2021 #88721

    My wife baited me into going to a freedom rally at our statehouse recently. She said there would be lots of nice looking nurses protesting the mandates. It was a beautiful day and flirting was easy. The protest was peaceful, though there was one guy there that had attached his American flag to a Louisville Slugger. Nice touch. I did get the opportunity to write a note to the governor and personally deliver it to his office.

    I have been reading Walter Chestnut’s takes on the spike for a while. Don’t get me wrong, it is his ice and he can do triple axles around me all day long but it gets a little exasperating with all the sciencey dot connecting, whose to say if it he is right or wrong. Then again what isn’t the spike capable of? This last one though might be a nerdy attempt get a date with Nikki Manaj.

    Did you see the Malone/Bossche interview with Dr. Phillips that germ posted recently? Bossche speaks and then Phillips looks to Malone to translate into caveman speak for the rest of us. The funny part was Malone has been letting his beard grow and is looking a bit like a caveman these days.

    @Doc Robinson,
    What if the spike is not pathogenic and we have simply projected our collective pathological fear and ignorance on it?

    What if the mRNA injections down regulate the immune system so that these surface glycoproteins aren’t seen as a threat. Actually, our immune system isn’t even alerted until there is enough of them and our defenses are down so they can mount an attack. Without the machinery for viral replication. Why would our immune system take issue with them or even care?

    Another way to say it is that if I get the infection naturally my immune system is going to be like “hey I need to be on the look out for those glycoproteins on that sneaky devil”. But if I get the injection and there is really nothing pathogenic about the glycoproteins in and of themselves, then my immune my system will be lulled into a false sense of security when I encounter it or something like it in the future and a much higher viral load will be able to accumulate in my system. Isn’t this what we are seeing in the injected?

    Meet the spike.

    “The coronavirus sports a luxurious sugar coat. “It’s striking,” thought Rommie Amaro, staring at her computer simulation of one of the trademark spike proteins of SARS-CoV-2, which stick out from the virus’s surface. It was swathed in sugar molecules, known as glycans.

    “When you see it with all the glycans, it’s almost unrecognizable,” says Amaro, a computational biophysical chemist at the University of California, San Diego.

    Many viruses have glycans covering their outer proteins, camouflaging them from the human immune system like a wolf in sheep’s clothing.”

    So if we are only injecting the “sugar coat” and not the the whole enchilada, then how is that the immune system sees nothing more than a sheep?

    In installment #4, I posted a TEDx video by professor Bertozzi where she discusses how these “sugar coated” cells communicate with our immune system. How they can literally put our immune cells to sleep. Today in the Project Veritas video that Raul posted today. There is a screen capture of the J&J scientist Justin (I have no moral compass) Durrant text that said the following.

    “What I said about the formulations and cancer needs to stay between us. “

    Debt Rattle September 23 2021

    Anyways, I’m beginning to see a light at the end of the tunnel on how the spike leads to clotting in both the disease and the injection. But many times It’s not the right tunnel. We will see.

    For those that have read this far, leave you with this throw back.

    in reply to: Debt Rattle September 27 2021 #88355

    my parents,

    They are creating a list of all who read and shared that one so they know who to round up. Just kidding, still not Australia.

    Remember when the space shuttle burned up on reentry. It was due to damaged tiles that shield the vehicle from the extreme surface heating on reentry. The launch video captured pieces foam insulation hitting the shuttle. It was dismissed at first because it was foam. Eventually they built a cannon to fire a piece of foam of similar size and velocity. It damage the tiles.

    In the moment, everyone has bought into the idea that the spike protein is pathogenic once it enters the blood stream. What if it isn’t?

    in reply to: Debt Rattle September 27 2021 #88350

    @Doc Robinson,

    Diameter of a typical (human) capillary is 0.001mm (Area=785,714 nm^2)

    mRNA LNP diameter = 90-140nm (Average Area=10,391 nm^2)

    Outer Diameter of a 25 gauge needle=0.515mm (500X the capillary diameter)

    roughly 70 LNP can occupy the cross sectional area of a capillary.

    Red Blood Cells move thru capillaries single file.

    At any given moment, only about 5-10% of our capillary beds have blood flowing through them.

    in reply to: Debt Rattle September 27 2021 #88346

    @Doc Robinson,

    If injected IV, what type of cell(s) would be used would be used to build out a version with the spike protein?

    in reply to: Debt Rattle September 27 2021 #88342

    650K reads / 1500 comments for Spartacus letter on ZH. Mostly affirmative comments and noise. A few contrarian ones of note.

    “If SARS-CoV-2 is a virus similar to SARS it can not be a “blood disease”. A respiratory virus can not be a blood borne pathogen. It would also be nearly impossible to transmit through respiratory droplets. It is either a respiratory disease or it is not.”

    “It is like “coronavirus” is the do-everything, pixie dust of the virus world. Like the symptoms range from the sniffles to your internal organs imploding……..but there’s “natural immunity”?”

    “Funny how it “eats your blood vessels away” but the survival rate is 99.7%. Funny how it is a deadly bioweapon that is the greatest pandemic the world has ever seen, but if you are under 65 you have a better chance of dying from lightning strike.”

    This entire “letter” is a thumbnail description of this psyop…….right down to its anonymity and choice of pseudonym.

    in reply to: Debt Rattle September 27 2021 #88248

    Re: “We are all cattle now”

    I wonder if IBV was a thing when chickens could socially distance.

    in reply to: Spartacus #88225

    Body length=virus cell body diameter

    in reply to: Spartacus #88224

    Hmmm, has a Q-like vibe. Might be trying to punk us.

    Spartacus 1st summary point:

    “COVID-19 is a blood and blood vessel disease. SARS-CoV-2 infects the lining of human blood vessels, causing them to leak into the lungs.”

    This where my understanding is currently.

    On one side, we have the virus with all replicating machinery covered with surface spike glycoproteins that have a receptor binding domain that can attach to a cell expressing the ACE2.

    On the other side, we have injection that can create a non replicating version of this virus with spike glycoproteins on the surface which in theory can mimic the receptor binding domain properties of the spike glycoprotein on the virus.

    Endothelial make up our vascular plumbing and can express this ACE2 receptor. This could be bad if the replicating virus where to attach to this receptor.

    What does it mean if the non replicating version were to make it into the vascular system? With no replicating machinery, how does it damage the endothelial cell? Seems like a nothing burger in this case.

    In a live person (not so close to death) blood travels in the heart around 25 cm/s (10 in/s) on average. The diameter of the virus is on the order of 100nm. In my math is right, then virus would be moving 2.5 million body lengths every second. In the moment, I have a hard time believing that the small patch in the spike receptor binding domain has any chance of attaching to an ACE2 receptor in the heart.

    Here are the similarities between the respiratory infection and the mRNA injection. The infection attacks the epithelial cells in the air sacs ultimately gaining access to the nearby capillaries.

    In the intramuscular injection, non replicating versions of the virus are created and somehow gain access to nearby capillaries.

    I am working on putting a floor under my working theory. The links that Raul and Germ have posted over the last few days have helped fill in some gaps that I was wrestling with. And this cancer stuff, holy heck. It’s been fascinating path of discovery overall, even if I come up bagel.

    in reply to: Debt Rattle September 24 2021 #87985

    Raul, thanks again for the line up today.

    In the limit, as we provide opportunities for the virus to optimize it’s symbiotic relationship with our immune system we are likely to live long miserable immunocompromised lives, irrespective of injection status. Until, of course, some of the sickly survivors mate up produce a gene in their offspring that provides protection.

    in reply to: Debt Rattle September 23 2021 #87947

    Spikes are Bad and Spikes in the Blood are Really Bad (installment 4 of ??)

    Here is my current working theory.

    The virus will attach to a host cell that has an ACE2 receptor using is surface spike protein. Once inside the host cell, hidden from the immune system, it will make many copies of itself. The host cell dies and now there are many new copies of the virus available to attach to more host cells. Cells with ACE2 receptors are found throughout the body.


    The spike protein is also capable of binding to Sialic Acid (SA). The blood is a target rich environment for SA. Red blood cells (RBCs) are coated with it. Once attached to an RBC, it can move throughout the body until conditions are favorable for it to attach to a call expressing ACE2. The affinity for the spike to bind to ACE2 is much greater than SA. Fluidic forces, like turbulence, could free the virus from the RBC, possibly damaging it. Also in regions of slow blood flow and high virus count, RBCs and the viruses could clump up due to the SA binding properties. This would impede blood flowing in that region ultimately causing vascular damage.

    Though this video is about cancer, there is enlightening info about SA.

    I first started to consider the role of SA in a Covid infection after seeing the figure below. These are some of the viruses that are in the corona virus family. It shows the different types of host receptors for binding. Also, shown is the growing affinity for SA going from left to right.


    The corresponding paper is linked here.

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