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 March 1, 2022  Posted by at 12:23 pm Finance Tagged with: , , , , , , , ,  11 Responses »


Edward Hopper Approaching a city 1946

 

 

On September 26, 2021, I posted the Spartacus letter in its entirety, because I thought it was too good to “disappear” in our daily news aggregator, Debt Rattle. This turned out to be a good decision, since the letter vanished from its original server soon afterward. But it was -and is- still here. Zero Hedge reposted my repost, and things went off from there, generating some 200,000 views here and 1.85 million at Zero Hedge. Plus who knows how many at other places.

The Spartacus collective -we now know they are indeed a group- returned a few days ago with an update letter, now on Substack. That is probably a safer place then their original Docdroid site, but I’ll repost this one as well. Nice to see they are aware of the Automatic Earth’s role in garnering attention for the first letter. My pleasure. You guys are very good.

Note: the original, COVID-19: A Web of Corruption, is here.

 

 

Foreword

My name is Spartacus, and I ve had enough.

I am one of the authors of the Spartacus Letter, a document that took the world by storm in September of 2021.

To date, four versions of the letter have been published, and all four can be viewed here:

Spartacus Letter V1

Spartacus Letter V2

Spartacus Letter V3

Spartacus Letter V4

We shared this document with numerous news outlets and sent it directly to Dr. Robert Malone, who linked it on his Twitter account. From there, it was reposted on the Automatic Earth blog, and then, on ZeroHedge, where it garnered over a million hits.

It was quickly decried as misinformation both by freelance analysts on Twitter, and by fact-checkers:

Twitter avatar for @conspirator0Conspirador NorteOo @conspirator0

The “Spartacus” COVID disinfo document first appeared on docdroid(dot)com on Sept 24 2021. The first Twitter account to share it was a small Italian-language account, @number229401056. The second was @RWMaloneMD, whose tweet linking the letter was retweeted nearly 1000 times.

Image

If you perform a Google search for the Spartacus Letter, Google s algorithm prioritizes one result above all others: a fact-check by Newswise debunking it, which, unsurprisingly, 77% of visitors to the page disagree with.

Is this the case? Could ICENI, with a fully-sourced document with over 600 citations that lays out an extensive argument regarding the nature and origins of COVID-19, have been intending to deliberately mislead the public, as our detractors state?

Perhaps we were not explicit enough.

SARS-CoV-2 is manmade. It was produced at the Wuhan Institute of Virology with US Government funding. It is the lynchpin of a plan by ruthless elites to take over the entire world. There is enough circumstantial evidence that, if taken together, implicates numerous government officials and scientists in this plot.

The people involved could be charged with crimes against humanity for their complicity in this, but if they were, it would bring the entire system crashing down around our ears simply due to the sheer number of the guilty and the loftiness of their offices.

There is no way to sugarcoat any of this. If people remain ignorant of the nature of COVID-19 and the events leading up to the present, then the petty tyrants stripping us of our civil liberties will win.

What follows will be something of a recapitulation of the Spartacus Letter, in great detail, with numerous hyperlinks and small blocks of quoted material and images within fair use limitations, for the sake of commentary and criticism of public figures.

What is COVID-19?

COVID-19 is the disease caused by SARS-CoV-2, a close relative of SARS-CoV, the causative agent of Severe Acute Respiratory Syndrome, also known as SARS. SARS-CoV-2 is a coronavirus of the betacoronavirus genus, which it shares with SARS-CoV and MERS-CoV.

The healthcare establishment and the media have misrepresented COVID-19 as a lower respiratory disease, as a form of pneumonia. They have done so consistently for the past two years.

This time, Cavuto said he had a “far, far more serious strand” because his “very compromised immune system” simply hasn’t benefited in the same way from the vaccines as those with healthy immune systems.

Cavuto said that his most recent case of Covid-19 had led to pneumonia and landed him “in intensive care for quite a while.”

While it is certainly true that COVID-19 can cause pneumonia, it would be inaccurate to describe COVID-19 as a pneumonia per se. Due to the significant expression of ACE2 in vascular endothelial cells and pericytes as part of the Renin-Angiotensin-Aldosterone System (RAAS), SARS-CoV-2 preferentially attacks the lining of blood vessels and small capillaries, severely inflaming them, leading to sepsis, capillary leak, pulmonary edema, and yes, pneumonia.

However, therein lies the crucial distinction: SARS-CoV-2 injures the lungs by infecting the blood vessels that supply the alveoli and triggering sepsis and ARDS. It would be more accurate, therefore, to describe COVID-19 not as a pneumonia, but as a disease of the circulatory system.

This has been known since April of 2020, when University Hospital Zurich proclaimed that COVID-19 was, in actuality, a vascular endotheliitis.

During analyses of tissue samples taken from deceased COVID-19 patients taken following an autopsy, pathologists at University Hospital Zurich have now discovered that patients are not just suffering from an inflammation of the lungs, but also from an inflammation of all endothelial tissue in a wide range of organs. In addition, pathologist Prof. Zsuzsanna Varga has been able to use an electron microscope to verify for the first time that SARS-CoV-2 is present and causes cell necrosis in endothelial tissue.

The nature of COVID-19 as a vascular endotheliitis is now well-established in the primary literature.

The Lancet – Endothelial cell infection and endotheliitis in COVID-19

Post-mortem analysis of the transplanted kidney by electron microscopy revealed viral inclusion structures in endothelial cells (figure A, B). In histological analyses, we found an accumulation of inflammatory cells associated with endothelium, as well as apoptotic bodies, in the heart, the small bowel (figure C) and lung (figure D). An accumulation of mononuclear cells was found in the lung, and most small lung vessels appeared congested.

European Heart Journal – COVID-19 is, in the end, an endothelial disease

Inflammatory activation of endothelial cells can disrupt VE-cadherin largely responsible for the integrity of the endothelial barrier function.62 Activated endothelial cells can also express matrix metalloproteinases that can degrade the basement membrane and further interrupt endothelial barrier function. In small vessels, such as those that embrace alveoli in the lung, this impaired barrier function can lead to capillary leak.

What these papers are essentially describing is endothelial dysfunction and endothelial injury in the context of acute sepsis. Researchers have stated that severe COVID-19 is a form of sepsis. Why is this important? Because it has serious implications for how COVID-19 may be successfully treated. It also explains why many current treatments fail to rescue critically-ill patients.

Acute sepsis does a number of terrible things to the circulatory system and vital organs. One thing it does is severely disrupt the balance of oxidation and reduction reactions in the body.

Redox Report – Sepsis, oxidative stress, and hypoxia: Are there clues to better treatment?

Sepsis is a clinical syndrome characterized by systemic inflammation, usually in response to infection. The signs and symptoms are very similar to Systemic Inflammatory Response Syndrome (SIRS), which typically occur consequent to trauma and auto-immune diseases. Common treatments of sepsis include administration of antibiotics and oxygen. Oxygen is administered due to ischemia in tissues, which results in the production of free radicals. Poor utilization of oxygen by the mitochondrial electron transport chain can increase oxidative stress during ischemia and exacerbate the severity and outcome in septic patients. This course of treatment virtually mimics the conditions seen in ischemia reperfusion disorders. Therefore, this review proposes that the mechanism of free radical production seen in sepsis and SIRS is identical to the oxidative stress seen in ischemia reperfusion injury. Specifically, this is due to a biochemical mechanism within the mitochondria where the oxidation of succinate to fumarate by succinate dehydrogenase (complex II) is reversed in sepsis (hypoxia), leading to succinate accumulation. Oxygen administration (equivalent to reperfusion) rapidly oxidizes the accumulated succinate, leading to the generation of large amounts of superoxide radical and other free radical species. Organ damage possibly leading to multi-organ failure could result from this oxidative burst seen in sepsis and SIRS. Accordingly, we postulate that temporal administration with anti-oxidants targeting the mitochondria and/or succinate dehydrogenase inhibitors could be beneficial in sepsis and SIRS patients.

Another thing it does is promote endothelial dysfunction.

International Journal of Molecular Sciences – Endothelial Dysfunction and Neutrophil Degranulation as Central Events in Sepsis Physiopathology

Sepsis produces endothelial dysfunction, forcing a pro-adhesive, procoagulant and antifibrinolytic state in endothelial cells, thus altering the hemostasis, leucocyte trafficking, inflammation, barrier function and microcirculation [23].

We know this is occurring in COVID-19, because COVID-19 sufferers have elevated levels of both inflammatory cytokines such as TNF-a and IL-6, and oxidative and nitrosative stress biomarkers such as nitrotyrosine, coupled with low nitric oxide bioavailability. This has led some to hypothesize that, ultimately, severe COVID-19 is death by neutrophilia .

International Journal of Biological Sciences – A Multiple-Hit Hypothesis Involving Reactive Oxygen Species and Myeloperoxidase Explains Clinical Deterioration and Fatality in COVID-19

Multi-system involvement and rapid clinical deterioration are hallmarks of coronavirus disease 2019 (COVID-19) related mortality. The unique clinical phenomena in severe COVID-19 can be perplexing, and they include disproportionately severe hypoxemia relative to lung alveolar-parenchymal pathology and rapid clinical deterioration, with poor response to O2 supplementation, despite preserved lung mechanics. Factors such as microvascular injury, thromboembolism, pulmonary hypertension, and alteration in hemoglobin structure and function could play important roles. Overwhelming immune response associated with cytokine storms could activate reactive oxygen species (ROS), which may result in consumption of nitric oxide (NO), a critical vasodilation regulator. In other inflammatory infections, activated neutrophils are known to release myeloperoxidase (MPO) in a natural immune response, which contributes to production of hypochlorous acid (HOCl). However, during overwhelming inflammation, HOCl competes with O2 at heme binding sites, decreasing O2 saturation. Moreover, HOCl contributes to several oxidative reactions, including hemoglobin-heme iron oxidation, heme destruction, and subsequent release of free iron, which mediates toxic tissue injury through additional generation of ROS and NO consumption. Connecting these reactions in a multi-hit model can explain generalized tissue damage, vasoconstriction, severe hypoxia, and precipitous clinical deterioration in critically ill COVID-19 patients. Understanding these mechanisms is critical to develop therapeutic strategies to combat COVID-19.

This is also known as respiratory burst, or neutrophil degranulation, or, in extreme cases, neutrophil extracellular trap formation:

Neutrophils use enzymes such as superoxide dismutase (SOD) and myeloperoxidase (MPO) to produce hydrogen peroxide and hypochlorous acid (peroxide and bleach, essentially) in order to destroy bacteria and other pathogens by attacking their membranes with powerful oxidants.

Normally, our cells, which are made of essentially the same stuff as bacteria, survive this by using powerful antioxidant enzymes such as glutathione peroxidase (GPX) to break down hydrogen peroxide into water and reduce harmful lipid hydroperoxides to their corresponding alcohols.

ScienceDirect – Glutathione Peroxidase

Unfortunately, this virus has a very nasty trick up its sleeve. SARS-CoV-2 can directly inhibit the Nrf2 pathway, blocking endogenous antioxidant enzymes from functioning correctly.

Nature – SARS-CoV2-mediated suppression of NRF2-signaling reveals potent antiviral and anti-inflammatory activity of 4-octyl-itaconate and dimethyl fumarate

To identify host factors or pathways important in the control of SARS-CoV2 infection, publicly available transcriptome data sets including transcriptome analysis of lung biopsies from COVID-19 patients were analyzed using differential expression analysis14. Here, genes linked with inflammatory and antiviral pathways, including RIG-I receptor and Toll-like receptor signaling, were enriched in COVID-19 patient samples, whereas genes associated with the NRF2 dependent antioxidant response were suppressed in the same patients (Fig. 1a c). That NRF2-induced genes are repressed during SARS-CoV2 infections was supported by reanalysis of another data-set building on transcriptome analysis of lung autopsies obtained from five individual COVID-19 patients (Desai et al.15) (Fig. 1d). Furthermore, that the NRF2-pathway is repressed during infection with SARS-CoV2 was supported by in vitro experiments where the expression of NRF2-inducible proteins Heme Oxygenase 1 (HO-1) and NAD(P)H quinone oxydoreducatse 1 (NqO1) was repressed in SARS-CoV2 infected Vero hTMPRSS2 cells while the expression of canonical antiviral transcription factors such as STAT1 and IRF3 were unaffected (Supplementary Fig. 1). These data indicate that SARS-CoV2 targets the NRF2 antioxidant pathway and thus suggests that the NRF2 pathway restricts SARS-CoV2 replication.

The Nrf2 pathway directly regulates the function of glutathione peroxidase.

Elsevier – Nrf2-regulated glutathione recycling independent of biosynthesis is critical for cell survival during oxidative stress

Nuclear factor-erythroid 2 p45-related factor 2 (Nrf2) is the primary transcription factor protecting cells from oxidative stress by regulating cytoprotective genes, including the antioxidant glutathione (GSH) pathway. GSH maintains cellular redox status and affects redox signaling, cell proliferation, and death. GSH homeostasis is regulated by de novo synthesis as well as GSH redox state; previous studies have demonstrated that Nrf2 regulates GSH homeostasis by affecting de novo synthesis.

This is, of course, why severely ill COVID-19 patients are noted to have glutathione deficiencies.

Antioxidants – Severe Glutathione Deficiency, Oxidative Stress and Oxidant Damage in Adults Hospitalized with COVID-19: Implications for GlyNAC (Glycine and N-Acetylcysteine) Supplementation

Humanity is battling a respiratory pandemic pneumonia named COVID-19 which has resulted in millions of hospitalizations and deaths. COVID-19 exacerbations occur in waves that continually challenge healthcare systems globally. Therefore, there is an urgent need to understand all mechanisms by which COVID-19 results in health deterioration to facilitate the development of protective strategies. Oxidative stress (OxS) is a harmful condition caused by excess reactive-oxygen species (ROS) and is normally neutralized by antioxidants among which Glutathione (GSH) is the most abundant. GSH deficiency results in amplified OxS due to compromised antioxidant defenses. Because little is known about GSH or OxS in COVID-19 infection, we measured GSH, TBARS (a marker of OxS) and F2-isoprostane (marker of oxidant damage) concentrations in 60 adult patients hospitalized with COVID-19. Compared to uninfected controls, COVID-19 patients of all age groups had severe GSH deficiency, increased OxS and elevated oxidant damage which worsened with advancing age. These defects were also present in younger age groups, where they do not normally occur. Because GlyNAC (combination of glycine and N-acetylcysteine) supplementation has been shown in clinical trials to rapidly improve GSH deficiency, OxS and oxidant damage, GlyNAC supplementation has implications for combating these defects in COVID-19 infected patients and warrants urgent investigation.

Glutathione deficiencies, endothelial dysfunction, and chronic oxidative stress all point essentially to the same thing: untreated chronic malnutrition brought on by consumption of an energy-rich, micronutrient-poor diet. This is also known as Metabolic Syndrome, and has a close association with endothelial dysfunction and, indeed, premature aging of the blood vessels. It is, of course, why COVID-19 causes more severe illness in those with diabetes, high blood pressure, obesity, and old age. All of these conditions involve pre-existing endothelial dysfunction that renders one more vulnerable to sepsis.

There is, in fact, a close association between the severity of COVID-19 and the quality of one s diet.

BMJ – Diet quality and risk and severity of COVID-19: a prospective cohort study

Over 3 886 274 person-months of follow-up, 31 815 COVID-19 cases were documented. Compared with individuals in the lowest quartile of the diet score, high diet quality was associated with lower risk of COVID-19 (HR 0.91; 95% CI 0.88 to 0.94) and severe COVID-19 (HR 0.59; 95% CI 0.47 to 0.74). The joint association of low diet quality and increased deprivation on COVID-19 risk was higher than the sum of the risk associated with each factor alone (Pinteraction=0.005). The corresponding absolute excess rate per 10 000 person/months for lowest vs highest quartile of diet score was 22.5 (95% CI 18.8 to 26.3) among persons living in areas with low deprivation and 40.8 (95% CI 31.7 to 49.8) among persons living in areas with high deprivation.

It is plausible, though not conclusively proven, that COVID-19 could be warded off by simple changes to diet and exercise habits, especially the inclusion of antioxidant-rich foods high in Vitamin D, cysteine, dietary nitrate, and selenium in one s diet.

Rather than suggesting that people eat better and go on jogs, the authorities have spent the past two years locking people down, which has made them both more obese and more vulnerable to the virus.

Wiley – Effects of COVID-19 lockdown on eating disorders and obesity: A systematic review and meta-analysis

A total of 26 studies met inclusion criteria (n = 3399, 85.7% female). The pooled prevalence of symptomatic deterioration in EDs was 65% (95% CI[48,81], k = 10). The pooled prevalence of increased weight in obesity was 52% (95% CI[25,78], k = 4). More than half of the participants experienced depression and anxiety. Moreover, at least 75% of the individuals with EDs reported shape and eating concerns, and increased thinking about exercising.

What is actually happening in COVID-19 that causes all this oxidative damage? First, superoxide reacts with nitric oxide to form peroxynitrite, a damaging nitrogen radical. Then, peroxynitrite reacts with tetrahydrobiopterin in endothelial nitric oxide synthase, leading to those enzymes becoming uncoupled , which makes them recursively produce superoxide in a damaging biological feedback loop. Dr. Martin L. Pall refers to this phenomenon by the moniker NO/ONOO- Disease .

International Journal of Molecular Sciences – The NO/ONOO-Cycle as the Central Cause of Heart Failure

The NO/ONOO-cycle is a primarily local, biochemical vicious cycle mechanism, centered on elevated peroxynitrite and oxidative stress, but also involving 10 additional elements: NF-∫B, inflammatory cytokines, iNOS, nitric oxide (NO), superoxide, mitochondrial dysfunction (lowered energy charge, ATP), NMDA activity, intracellular Ca2+, TRP receptors and tetrahydrobiopterin depletion. All 12 of these elements have causal roles in heart failure (HF) and each is linked through a total of 87 studies to specific correlates of HF. Two apparent causal factors of HF, RhoA and endothelin-1, each act as tissue-limited cycle elements. Nineteen stressors that initiate cases of HF, each act to raise multiple cycle elements, potentially initiating the cycle in this way. Different types of HF, left vs. right ventricular HF, with or without arrhythmia, etc., may differ from one another in the regions of the myocardium most impacted by the cycle. None of the elements of the cycle or the mechanisms linking them are original, but they collectively produce the robust nature of the NO/ONOO-cycle which creates a major challenge for treatment of HF or other proposed NO/ONOO-cycle diseases. Elevated peroxynitrite/NO ratio and consequent oxidative stress are essential to both HF and the NO/ONOO-cycle.

As nitric oxide levels decrease and superoxide predominates (a textbook phenomenon in endothelial dysfunction), superoxide dismutase makes hydrogen peroxide, and then myeloperoxidase makes hypochlorous acid. Hypochlorous acid strips iron from heme. Then, free unliganded iron, hydrogen peroxide, and superoxide react in the Haber-Weiss and Fenton reactions to form damaging hydroxyl radicals.

Arch Neurosci – Oxidative Stress Gated by Fenton and Haber Weiss Reactions and Its Association With Alzheimer s Disease

It is difficult for most people to comprehend just how damaging hydroxyl radicals can be. Hydroxyl radicals occur naturally in the upper atmosphere, where they destroy pollutants. When they occur in the body, they oxidize lipids and DNA instantaneously, within nanoseconds, and no enzyme exists that can detoxify them. Hydroxyl radicals are often produced on purpose with hydrogen peroxide and iron catalyst in hydroxyl generators to create a powerful oxidant concoction that decontaminates and bleaches wastewater streams and HVAC systems by rapidly destroying biological material.

How fast?

That fast.

Why are COVID-19 patients dying in droves when they re intubated, with mortality from mechanical ventilation approaching 97% in some cases? It s because intubation mimics the physiology of ischemia-reperfusion injury. Under the acute sepsis triggered by COVID-19, cells experience hypoxia. They become stressed and switch to anaerobic metabolism and glycolysis to make ATP as a desperate last resort. Then, these cells are suddenly fed with O2 by a ventilator, which causes them to switch back to aerobic metabolism. As this happens, hypoxanthine and succinate breakdown produces superoxide radicals in very large amounts.

Superoxide is a precursor to many other types of radicals, as described above. There s even a name for it; the kindling radical/bonfire hypothesis .

Springer – Vascular Redox Signaling, Redox Switches in Endothelial Nitric Oxide Synthase (eNOS Uncoupling), and Endothelial Dysfunction

Many diseases and drug-induced complications are associated with or even caused by an imbalance between the formation of reactive oxygen and nitrogen species (RONS) and antioxidant enzymes catalyzing the breakdown of these harmful oxidants. According to the kindling radical hypothesis, initial formation of RONS may trigger the activation of additional sources of RONS in certain pathological conditions.

This process of ROS release greatly accelerates the damage caused by the virus, promoting lipid peroxidation and the formation of damage-associated molecular patterns and oxidation-specific epitopes. The DAMPs summon more neutrophils by their interaction with PRRs, which release more damaging enzymes. The OSEs cause the body to form autoantibodies against oxidized lipids, somewhat similar to some aspects of the pathophysiology of Lupus.

AHA Journals – Oxidation-Specific Epitopes Are Danger-Associated Molecular Patterns Recognized by Pattern Recognition Receptors of Innate Immunity

Oxidation reactions are vital parts of metabolism and signal transduction. However, they also produce reactive oxygen species, which damage lipids, proteins and DNA, generating oxidation-specific epitopes. In this review, we will discuss the hypothesis that such common oxidation-specific epitopes are a major target of innate immunity, recognized by a variety of pattern recognition receptors (PRRs). By analogy with microbial pathogen associated molecular patterns (PAMPs), we postulate that host-derived, oxidation-specific epitopes can be considered to represent danger (or damage) associated molecular patterns (DAMPs). We also argue that oxidation-specific epitopes present on apoptotic cells and their cellular debris provided the primary evolutionary pressure for the selection of such PRRs. Further, because many PAMPs on microbes share molecular identity and/or mimicry with oxidation-specific epitopes, such PAMPs provided a strong secondary selecting pressure for the same set of oxidation-specific PRRs as well.

This severe oxidative stress promotes steroid insensitivity. Suddenly, the corticosteroids stop working and the patient experiences inflammatory rebound.

Antioxidants – Oxidative Stress Promotes Corticosteroid Insensitivity in Asthma and COPD

Reactive oxygen and nitrogen species (RONS) promote corticosteroid insensitivity by disrupting glucocorticoid receptor (GR) signaling, leading to the sustained activation of pro-inflammatory pathways in immune and airway structural cells.

Elsevier – Steroid resistance and rebound phenomena in patients with COVID-19

A total of 319 COVID-19 patients were admitted to our hospital and 113 patients met inclusion criteria. The success group had 83 patients (73.5%), the rebound group had nine patients (8.0%), and the refractory group had 21 patients (18.6%). Compared with the success group, the rebound group received corticosteroids earlier, for a shorter duration, and stopped them sooner. The median time from symptom onset to rebound was 12 days. There was no rebound after 20 days. Compared with the success group, the hazard ratio for the number of days from corticosteroid onset to an improvement of two points on a seven-point ordinal scale was 0.29 (95% confidence interval [CI], 0.14 0.60, P < .001) for the rebound group versus 0.13 (95% CI, 0.07 0.25, P < .001) for the refractory group.

Antivirals such as Remdesivir, Kaletra, Ivermectin, and Hydroxychloroquine do nothing to stop this, because by the time someone is in the ER complaining of severe COVID-19 symptoms (which are actually acute sepsis brought on by a deranged, overreacting innate immune system), the virus is already gone.

Viruses – Remdesivir for Early COVID-19 Treatment of High-Risk Individuals Prior to or at Early Disease Onset Lessons Learned

Similarly to IAV infection, the highest viral load and infectivity for SARS-CoV-2 are observed +/”1 day around the day of symptom onset [15]. Both the amount of infectious virus as well as the amount of viral RNA as measured by qRT-PCR decrease rapidly thereafter. Accordingly, the number of cells within the patient s respiratory tract that are newly infected with SARS-CoV-2 declines sharply within a few days of disease onset. It is now well accepted that immunopathology plays a key role in severe COVID-19 [16]. Accordingly, treatment with corticosteroids, such as dexamethasone, improves survival in critically ill COVID-19 patients in the later stages of the disease [17].

Ultimately, the afflicted cells begin dying of ferroptosis and parthanatos.

PubMed – Ferroptosis: mechanisms, biology, and role in disease

As GPX4 is the major PLOOH-neutralizing enzyme, a general mechanism underlying erastin/RSL3-induced ferroptosis emerged: both compounds inactivate GPX4 RSL3 does so directly, and erastin does so indirectly by inhibiting cystine import, thus depriving cells of cysteine, an essential cellular antioxidant and a building block of GSH. Consequently, PLOOHs accumulate, possibly causing rapid and unrepairable damage of plasma membrane, leading to cell death (Fig. 2A). Conceptually, these findings establish ferroptosis as a cell death modality with mechanisms distinct from other known death processes. The pharmacological and genetic tools developed herein enable, and have become indispensable for, ferroptosis research.

PubMed – Parthanatos: mitochondrial-linked mechanisms and therapeutic opportunities

In the context of the CNS, which is highlighted in this review to illustrate PARP-1-mediated cell death mechanisms that are shared in most cases by non-neuronal systems, stimuli that induce pathological activation of PARP-1 in in vitro and in vivo studies include oxidative stress by reactive oxygen species (ROS), such as hydrogen peroxide (H2O2) or hydroxyl radical, nitrosative stress from NO or peroxynitrite (ONOO”), inflammation, ischaemia (or ischaemic reperfusion), hypoxia, hypoglycaemia and DNA-alkylating agents, such as N-methyl-N -nitro-N-nitrosoguanidine (MNNG).

That, in a nutshell, is the central pathophysiology of COVID-19. The virus has many other aspects. It can promote hypercoagulability and attack numerous vital organs throughout the body, including the brain, olfactory system, gastrointestinal system, pancreas, kidneys, liver, and even fat cells. However, all of these things occur in the context of acute sepsis and endothelial injury.

In short, COVID-19 is not the disease people have been told it is, and, as per ostracized physicians such as Dr. Peter McCullough, Dr. Paul E. Marik, and Dr. Vladimir Zelenko, it is not being treated correctly.

The medical establishment has shunned those pushing for time-sensitive early outpatient treatment of COVID-19 sepsis. The standard of care for COVID-19 is to send people home without a prescription for anything; no antivirals, no antioxidants. Either their infection resolves without incident, or they get sicker, come back, and are intubated and proned, their diaphragm paralyzed with drugs so they can t fight the ventilator, a drip of steroids running into their arm.

This is state-sponsored medical murder.

Why don t people want the vaccines?

Because they are extremely fishy even at first glance. That s why.

Even people with no knowledge of the background of all of this are deeply suspicious of the speed with which these vaccines were fast-tracked, as well as the authorities insistence on people taking them, on pain of job loss, being banned from travel, and essentially being evicted from society.

In absolute terms, COVID-19 is not even particularly lethal. The infection fatality rate for those under age 50 with no comorbidities is very low. In that context, the authorities panicked response and open hostility doesn t seem to make any sense at all.

The mRNA and viral vector vaccines for COVID-19 work essentially by using human cells as a bioreactor, delivering genetic material into cells to get them to express a modified form of SARS-CoV-2 Spike as a vaccine antigen. They do not contain whole virus, nor do they stimulate an antibody response against every structural protein of SARS-CoV-2.

It sounds almost elegant. Any virus could, conceivably, have a tailor-made vaccine produced against it in a matter of days by plugging the gene sequence for its structural proteins in and making a lipid nanoparticle or viral vector containing that genetic material.

Just one problem. It causes severe and life-threatening side effects.

Clinical Infectious Diseases – Intravenous Injection of Coronavirus Disease 2019 (COVID-19) mRNA Vaccine Can Induce Acute Myopericarditis in Mouse Model

Although significant weight loss and higher serum cytokine/chemokine levels were found in IM group at 1 2 days post-injection (dpi), only IV group developed histopathological changes of myopericarditis as evidenced by cardiomyocyte degeneration, apoptosis, and necrosis with adjacent inflammatory cell infiltration and calcific deposits on visceral pericardium, although evidence of coronary artery or other cardiac pathologies was absent. Serum troponin level was significantly higher in IV group. Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) spike antigen expression by immunostaining was occasionally found in infiltrating immune cells of the heart or injection site, in cardiomyocytes and intracardiac vascular endothelial cells, but not skeletal myocytes. The histological changes of myopericarditis after the first IV-priming dose persisted for 2 weeks and were markedly aggravated by a second IM- or IV-booster dose. Cardiac tissue mRNA expression of interleukin (IL)-1≤, interferon (IFN)-≤, IL-6, and tumor necrosis factor (TNF)-± increased significantly from 1 dpi to 2 dpi in the IV group but not the IM group, compatible with presence of myopericarditis in the IV group. Ballooning degeneration of hepatocytes was consistently found in the IV group. All other organs appeared normal.

Steve Kirsch, a vocal opponent of the vaccine mandates, has written on Substack about this very topic, as well as attorney Thomas Renz s leaked DMED data, which the DOD have been trying desperately to cover up.

Steve Kirsch’s newsletter
DMED data is explosive. Mainstream media has been ordered to ignore it.
Summary The medical database used by the US military shows a huge uptick in serious events in 2021. Only events caused by the vaccine (as noted by the uptick in VAERS reports for these symptoms) were elevated. The DoD has claimed the increase was because events in earlier years were under reported and they have corrected the error&

Read more

Elsevier – SARS-CoV-2 spike protein interactions with amyloidogenic proteins: Potential clues to neurodegeneration

The post-infection of COVID-19 includes a myriad of neurologic symptoms including neurodegeneration. Protein aggregation in brain can be considered as one of the important reasons behind the neurodegeneration. SARS-CoV-2 Spike S1 protein receptor binding domain (SARS-CoV-2 S1 RBD) binds to heparin and heparin binding proteins. Moreover, heparin binding accelerates the aggregation of the pathological amyloid proteins present in the brain. In this paper, we have shown that the SARS-CoV-2 S1 RBD binds to a number of aggregation-prone, heparin binding proteins including A≤, ±-synuclein, tau, prion, and TDP-43 RRM. These interactions suggests that the heparin-binding site on the S1 protein might assist the binding of amyloid proteins to the viral surface and thus could initiate aggregation of these proteins and finally leads to neurodegeneration in brain. The results will help us to prevent future outcomes of neurodegeneration by targeting this binding and aggregation process.

SARS CoV 2 Spike Impairs DNA Damage Repair and Inhibits V(D)J Recombination In Vitro

Severe acute respiratory syndrome coronavirus 2 (SARS CoV 2) has led to the coronavirus disease 2019 (COVID 19) pandemic, severely affecting public health and the global economy. Adaptive immunity plays a crucial role in fighting against SARS CoV 2 infection and directly influences the clinical outcomes of patients. Clinical studies have indicated that patients with severe COVID 19 exhibit delayed and weak adaptive immune responses; however, the mechanism by which SARS CoV 2 impedes adaptive immunity remains unclear. Here, by using an in vitro cell line, we report that the SARS CoV 2 spike protein significantly inhibits DNA damage repair, which is required for effective V(D)J recombination in adaptive immunity. Mechanistically, we found that the spike protein localizes in the nucleus and inhibits DNA damage repair by impeding key DNA repair protein BRCA1 and 53BP1 recruitment to the damage site. Our findings reveal a potential molecular mechanism by which the spike protein might impede adaptive immunity and underscore the potential side effects of full-length spike-based vaccines.

FDA ACCIDENTALLY DISPLAYS LIST OF COVID VACCINE SIDE EFFECTS INCLUDING DEATH, MYOCARDITIS AND AUTOIMMUNE DISEASE

Under no circumstances should people be compelled to take these experimental and highly harmful vaccines, period.

If that were all there was to this story, one could employ Hanlon s razor and chalk this all up as a series of very unfortunate medical blunders and not the bloodthirsty malice and contempt towards the public that it truly is.

However, this isn t even the half of it.

Those who dig deeper encounter a horror story of biblical proportions, involving private-public collusion, government malfeasance, and national security skullduggery at the highest levels.

Where did the virus come from?

Since the 2000s, DARPA has greatly expanded their biodefense portfolio. A fellow named Michael Callahan was involved in this work, among many others. His job was to study old Soviet biowarfare sites like the Vector Institute, looking for technologies that could be patented and commercialized.

Unlimited Hangout – DARPA s Man in Wuhan

Callahan s nose for business came into play early on in the pandemic. After studying data from over 6,000 patient records from Wuhan, he reportedly detected a pattern that could point to a possible treatment using a low-cost and widely available ingredient of an over-the-counter histamine-2 receptor antagonist called Famotidine , more commonly known as the brand name Pepcid.

As an aside, Famotidine is not just an antihistamine. It is also a powerful antioxidant that interrupts the Fenton reaction, which, if one understands the pathophysiology of COVID-19, means that Pepcid makes perfect sense as a therapeutic agent.

In 2010/2011, NIH, DARPA, and DTRA may have discovered a cure for almost all pandemic viruses, but rejected it, despite evidence of its efficacy in a murine model. It was called DRACO, or Double-Stranded RNA Activated Caspase Oligomerizer, and it was developed by an MIT scientist by the name of Todd Rider. It is a recombinant fusion protein that kills virally-infected cells by ordering them to undergo apoptosis if it detects viral dsRNA. In mouse experiments, it was shown to protect mice from influenza.

PLOS One – Broad-Spectrum Antiviral Therapeutics

Funding: This work is funded by grant AI057159 (http://www.niaid.nih.gov/Pages/default.aspx) from the National Institute of Allergy and Infectious Diseases and the New England Regional Center of Excellence for Biodefense and Emerging Infectious Diseases, with previous funding from the Defense Advanced Research Projects Agency, Defense Threat Reduction Agency, and Director of Defense Research & Engineering. The funders had no role in study design, data collection and analysis, decision to publish, or preparation of the manuscript. Opinions, interpretations, conclusions, and recommendations are those of the authors and are not necessarily endorsed by the United States government.

In the end, Dr. Rider had to resort to crowdfunding campaigns to continue his research. Unfortunately, these campaigns failed.

MEET TODD RIDER, THE MAN WHO MAYBE, PROBABLY CURED MOST OF THE VIRUSES ON EARTH

Modest amounts of funding from the National Institutes of Health have enabled the previous proof-of-concept experiments in cells and mice, but that funding grant is now over. Major pharmaceutical companies have the resources and expertise to carry new drugs like DRACO through the manufacturing scale-up, large-scale animal trials, and human trials required for FDA approval. However, before committing any of their own money, those companies want to see that DRACOs have already been shown to be effective against major clinically relevant viruses (such as members of the herpesvirus family), not just the proof-of-concept viruses (such as rhinovirus) that were previously funded by NIH. Thus the Valley of Death is the financial and experimental gap between the previously funded NIH proof-of-concept experiments and the threshold for convincing major pharmaceutical companies to advance DRACOs toward human trials.

However, I digress. Ever since the 2002/2003 SARS outbreak, there has been significant interest in SARS-CoV among virologists, including gain-of-function manipulation of SARS virions in the laboratory environment.

The usual stated purpose of gain-of-function research is to get ahead of pandemics by producing a human-adapted pathogen and preemptively vaccinating against it.

Potential Risks and Benefits of Gain-of-Function Research: Summary of a Workshop – Part 4

In the long-term it may also allow the generation of information that is not obtainable through other methods, but whether all the long-term benefits envisioned for GoF research will actually be realized is still unclear. Vaccine producers in particular disagree on whether GoF methods are essential for vaccine development, so the contributions of GoF research to vaccine development need careful evaluation. Increasing reliance on gene sequences to predict phenotypes may increase GoF research’s importance over time. As was clear from the presentations in Session 4 of the symposium, there is wide recognition that it is not yet possible to predict phenotype from genotype, but Dr. Philip Dormitzer, from Novartis Vaccines and a member of the symposium planning committee, noted that as more genotype-phenotype linkages are established, it may enable keeping certain viral characteristics out of vaccine strains.

Some scientists base their entire careers on this research, obtaining grant money from various institutions, including military think-tanks, to experiment on pathogens in this manner.

There s just one problem with this; gain-of-function research (a.k.a. Dual-Use Research of Concern ) has never successfully produced a vaccine against anything. It is simply bioweapon research by another, sanitized, euphemistic name.

GAIN-OF-FUNCTION RESEARCH ON SARS VIRUS, PERFORMED AT US LAB WITH BIO-CELLS FROM FORT DETRICK

Francis Boyle: We have an article here from the NAT MED 2015, December 21 SARS like cluster of circulating bat coronavirus show, potential for human emergence. This was at the University of North Carolina, in Chapel Hill. They have a biosafety lab level 3 there. And I have previously condemned them, for using gain-of-function work on MERS, which is the Middle East Respiratory Syndrome.

One of the foremost figures in coronavirus GOF research is a guy named Ralph Baric at UNC Chapel Hill.

He has been conducting research into various coronaviruses, including SARS, for many decades there. This is who Anthony Fauci is referring to when he speaks of GOF work being done in North Carolina .

‘Gain-of-function’ research: What it is and who is doing it in North Carolina

Gain-of-function research has been done in North Carolina, most notably at the lab of UNC-Chapel Hill researcher Ralph Baric. Baric is one of the world’s preeminent coronavirus researchers, beginning his study of the family of viruses in the 1990s before they were seen as potentially pandemic-level dangerous to humans. Baric has used gain-of-function techniques to show how coronaviruses could evolve to infect humans and to test new vaccine methods to neutralize them.

In 2015, Ralph Baric co-authored a paper with Shi Zhengli, a bat coronavirus expert from the Wuhan Institute of Virology, entitled A SARS-like cluster of circulating bat coronaviruses shows potential for human emergence .

The emergence of severe acute respiratory syndrome coronavirus (SARS-CoV) and Middle East respiratory syndrome (MERS)-CoV underscores the threat of cross-species transmission events leading to outbreaks in humans. Here we examine the disease potential of a SARS-like virus, SHC014-CoV, which is currently circulating in Chinese horseshoe bat populations1. Using the SARS-CoV reverse genetics system2, we generated and characterized a chimeric virus expressing the spike of bat coronavirus SHC014 in a mouse-adapted SARS-CoV backbone. The results indicate that group 2b viruses encoding the SHC014 spike in a wild-type backbone can efficiently use multiple orthologs of the SARS receptor human angiotensin converting enzyme II (ACE2), replicate efficiently in primary human airway cells and achieve in vitro titers equivalent to epidemic strains of SARS-CoV. Additionally, in vivo experiments demonstrate replication of the chimeric virus in mouse lung with notable pathogenesis. Evaluation of available SARS-based immune-therapeutic and prophylactic modalities revealed poor efficacy; both monoclonal antibody and vaccine approaches failed to neutralize and protect from infection with CoVs using the novel spike protein. On the basis of these findings, we synthetically re-derived an infectious full-length SHC014 recombinant virus and demonstrate robust viral replication both in vitro and in vivo. Our work suggests a potential risk of SARS-CoV re-emergence from viruses currently circulating in bat populations.

In other words, Ralph Baric and Shi Zhengli were colleagues. Anthony Fauci pointing to Ralph Baric at UNC Chapel Hill as evidence of the NIH not funding GOF research in Wuhan is downright farcical; these scientists collaborated openly on various projects over the past decade.

A year before this paper was published, a moratorium on US federal funding for gain-of-function research was put in place. This moratorium lasted from 2014 through 2017.

U.S. halts funding for new risky virus studies, calls for voluntary moratorium

A group calling itself the Cambridge Working Group issued a statement in July saying that studies with “potential pandemic pathogens” should be “curtailed” until the risks and benefits could be evaluated; it has garnered hundreds of signatures. Another group of scientists supporting the experiments they call themselves Scientists for Science defended the studies as safe but also called for a meeting to discuss the issues.

U.S. Government Gain-of-Function Deliberative Process and Research Funding Pause on Selected Gain-of-Function Research Involving Influenza, MERS, and SARS Viruses

New USG funding will not be released for gain-of-function research projects that may be reasonably anticipated to confer attributes to influenza, MERS, or SARS viruses such that the virus would have enhanced pathogenicity and/or transmissibility in mammals via the respiratory route. The research funding pause would not apply to characterization or testing of naturally occurring influenza, MERS, and SARS viruses, unless the tests are reasonably anticipated to increase transmissibility and/or pathogenicity.

Researchers whose careers depended on GOF research were openly hostile to the moratorium, despite the noble intent behind it of preventing a lab escape.

Researchers rail against moratorium on risky virus experiments

Andrew Hebbeler, assistant director for biological and chemical threats in the White House Office of Science and Technology Policy (OSTP), explained at a meeting today of the National Science Advisory Board for Biosecurity (NSABB) that the policy is a response to several recent biosafety lapses at federal labs involving mishandled samples of anthrax, H5N1, and smallpox. Although GOF actually encompasses “a huge swath of life sciences research,” he said, officials decided to focus only on influenza, MERS, and SARS because they are can be transmitted through the air and have the potential to spark a pandemic. OSTP told ScienceInsider that about two dozen studies funded by the National Institutes of Health (NIH) are affected; the pause also halts some studies at the U.S. Department of Agriculture.

In any case, the moratorium was ignored. Without contacting the White House for approval, federal funding for gain-of-function research continued, using intermediaries to subcontract the grants.

Enter Peter Daszak, the director of EcoHealth Alliance.

A man who pens psychotic love letters to viruses.

Springer – A Fall From Grace To& Virulence?

In Bruegel s painting of The Fall of the Rebel Angels we are witness to a tumbling maelstrom of falling rebel angels outcast from Heaven. Within the fray stands St. Michael in gilded armor, and his angels-at-arms serenely in pale albs, and almost as if threshing grain, hewing and striking down this inconceivable rout. The main focus of the image and what draws the eye is the extraordinarily creative mElange of creatures; mixtures of human, animal, plant, and inanimate objects slashing and stabbing as they fall from the great battlefields in the skies. They pour down in a vast column that stretches infinitely from the luminous sun; they fall from the light to the darkness. The column of falling angels is so numerous that it widens to encompass the whole lower canvas as it approaches the viewer. With a start, then, we realize that Bruegel intends that we too are in the thick of this. Will we succumb to the multitudinous horde? Are we to be cast downward into chthonic chaos represented here by the heaped up gibbering phantasmagory against which we rail and struggle?

Over the course of the past decade, EcoHealth Alliance have received millions of dollars in funding from NIH/NIAID, USAID (a known CIA front), and DTRA (yes, the Pentagon) to subcontract shady gain-of-function research to places like the Wuhan Institute of Virology. This can be confirmed by checking usaspending.gov and taking note of the exact amounts awarded.

Spending by Prime Award – EcoHealth Alliance

EcoHealth Alliance received millions of our tax dollars under UC Davis s PREDICT program, which is a part of USAID s EPT program.

UC Davis – PREDICT

PREDICT, a project of USAID s Emerging Pandemic Threats (EPT) program, was initiated in 2009 to strengthen global capacity for detection of viruses with pandemic potential that can move between animals and people. PREDICT has made significant contributions to strengthening global surveillance and laboratory diagnostic capabilities for both known and newly discovered viruses within several important virus groups, such as filoviruses (including ebolaviruses), influenza viruses, paramyxoviruses, and coronaviruses.

Independent researchers have done extensive digging into Daszak, uncovering incredibly alarming information about his background and character.

Fauci lied under oath and covered up for Daszak when he denied NIH never funded Gain-of-Threat research: Daszak authored a paper stating “This summary contains the information for the 2014 and 2017 NIH and NIAID grants to the Ecohealth Alliance that funded the WIV research on bat conronaviruses. As the grant description shows, this research included gain-of-function / gain-of-threat research to make coronaviruses viruses more pathogenic using techniques including genetic engineering, cell culture, and animal experimentation.”

DRASTIC Research uncovered documents showing that, in 2018, DARPA turned down a proposal by EcoHealth Alliance to receive grant money to conduct what they called the DEFUSE project, which EcoHealth had offered as a response to DARPA s PREEMPT program. The research would have involved exposing bats in caves to recombinant Spike proteins. DARPA, in their rejection letter, stated that this was dangerous gain-of-function research.

Moderna actually had considerable DARPA and BARDA funding for their mRNA technology, as a matter of fact.

mRNA Strategic Collaborators: Government Organizations

In October 2013, DARPA awarded Moderna up to approximately $25 million to research and develop potential mRNA medicines as a part of DARPA s Autonomous Diagnostics to Enable Prevention and Therapeutics, or ADEPT, program, which is focused on assisting with the development of technologies to rapidly identify and respond to threats posed by natural and engineered diseases and toxins. This award followed an initial award from DARPA given in March 2013. The DARPA awards have been deployed primarily in support of our vaccine and antibody programs to protect against Chikungunya infection.

This brings us to the start of the COVID-19 pandemic.

On December 12th, 2019, before anyone even knew an outbreak had occurred in Wuhan, Ralph Baric signed a material transfer agreement (seen on Page 105 of this document) to take delivery of mRNA coronavirus vaccine candidates developed and jointly-owned by NIAID and Moderna .

Wait, NIAID and Moderna co-own an mRNA vaccine? Who is the director of NIAID again?

Oh, right.

This was a whole month before China allegedly sent us the sequence to what would become known as SARS-CoV-2. Moderna claimed they made a vaccine from this sequence within 48 hours.

Moderna’s groundbreaking coronavirus vaccine was designed in just 2 days

On January 11, researchers from China published the genetic sequence of the coronavirus. Two days later, Moderna’s team and NIH scientists had finalized the targeted genetic sequence they would use in the vaccine.

Because, clearly, that isn t suspicious, or anything.

Ralph Baric also played a role in validating the use of Remdesivir in COVID-19.

Remdesivir, developed through a UNC-Chapel Hill partnership, proves effective against COVID-19 in NIAID human clinical trials

Remdesivir was developed through an academic-corporate partnership between Gilead Sciences and the Baric Lab at the University of North Carolina at Chapel Hill s Gillings School of Global Public Health. The biopharmaceutical company sought the talents of a research team led by William R. Kenan, Jr. Distinguished Professor of Epidemiology Ralph Baric, who has studied coronaviruses for more than 30 years and pioneered rapid-response approaches for the study of emerging viruses and the development of therapeutics.

Wuhan is, of course, host to China s only P4/BSL-4 virology lab, the Wuhan Institute of Virology. As a matter of fact, the P4 lab at the WIV was built with the help of Alain MErieux, the founder of bioMErieux, who offered his services to the CCP as a consultant for the endeavor.

The Wuhan lab at the core of a virus controversy

The 300 million yuan ($42 million) lab was completed in 2015, and finally opened in 2018, with the founder of a French bio-industrial firm, Alain Merieux, acting as a consultant in its construction.

Alain MErieux was presented an award for his collaboration with the Chinese Communist Party.

Alain MErieux receives the Prestigious Chinese Reform Friendship Award

Alain MErieux s award is a continuation of the longstanding relationship the MErieux family and its companies have built with China over the past 40 years. China has become a strategic location for all of Institut MErieux s work in the field of diagnostics, immunotherapy, and nutrition. Through its companies bioMErieux, Transgene, and MErieux Nutrisciences, and alongside the MErieux Foundation, Institut MErieux has partnered with Chinese authorities and health stakeholders to address major public health issues in the country.

Why is this such a big deal? Well, StEphane Bancel, the current CEO of Moderna, was formerly the CEO of bioMErieux.

Moderna CEO Worked Under Pharma Billionaire Who is Great Friends with Chinese Dictator Xi Jinping and Pushed for Wuhan Virology Lab

Moderna CEO Stephane Bancel, who is pushing experimental COVID-19 vaccines designed to alter the RNA of recipients, previously worked as CEO of BioMerieux, a firm owned by a billionaire who was instrumental in the development of the infamous virology lab in Wuhan, China.

By the time February, 2020 rolled around, the lab leak theory was gaining serious traction among alternative media sources. Peter Daszak was panicking, at this point. He authored a letter along with numerous scientists stating, unequivocally, that the WIV was not the source of the pandemic.

Statement in support of the scientists, public health professionals, and medical professionals of China combatting COVID-19

The rapid, open, and transparent sharing of data on this outbreak is now being threatened by rumours and misinformation around its origins. We stand together to strongly condemn conspiracy theories suggesting that COVID-19 does not have a natural origin. Scientists from multiple countries have published and analysed genomes of the causative agent, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2), and they overwhelmingly conclude that this coronavirus originated in wildlife, as have so many other emerging pathogens.

As his uncovered emails would later show, Daszak actually contacted Ralph Baric and instructed him not to sign the letter, fearing the conflict of interest it would entail, but not caring about his own, or that of the couple dozen scientists who did end up signing it.

Dr. Peter Daszak of EcoHealth Alliance Gets Brutal News Over Infamous Wuhan Conspiracy Theory Letter

Peter Daszak was also practically soiling himself at the prospect of all of this being traced back to USAID and UC Davis.

EcoHealth Alliance wanted to block disclosure of Covid-19-relevant virus data from China

As the outbreak spread to the US, Anthony Fauci waffled on masking, allegedly to prevent a run on masks, while the government turned down local offers by companies in the US to manufacture millions of masks.

Meanwhile, in New York, doctors cannot figure out why their patients on ventilators keep dying. Of course, it s because their patients have acute sepsis and the ventilators are making it worse by mimicking the pathophysiology of ischemia-reperfusion injury and accelerating COVID-19 s aggressive lipid peroxidation by feeding a ROS storm with O2, its main ingredient. They are setting off a deadly redox bomb in their patients chests by intubating them, but they don t realize this.

Of course, if a patient is blue in the face and desaturating, something must be done to alleviate it, but without endogenous glutathione peroxidase activity, increasing oxidative stress just makes this situation much, much worse. It actually increases hypoxia by chemically changing the blood and altering red blood cell morphology. ROS will compete with O2 for heme binding sites, crowding it out. Regardless of pneumonia or lung physiology or any of that, RBCs will become chemically incapable of accepting oxygen due to the simple laws of physics. Again, severe COVID-19 is an acute sepsis and endotheliitis first and second, and a pneumonia third, at best.

An anonymous scientist came up with a report that RaTG13 was a forgery, hand-entered into a BLAST database to falsify an ancestor for SARS-CoV-2, throwing suspicion off the WIV.

Sapan Desai and Surgisphere, a fake company, published a fake paper arguing that hydroxychloroquine caused heart rhythm irregularities. Before slamming Ivermectin as Horse Dewormer, the media called HCQ fish tank cleaner , drawing comparisons between it and the chemically similar chloroquine phosphate. Never mind that HCQ and chloroquine phosphate are essentially synthetic quinine, the main ingredient of tonic water and a historical malaria cure. Never mind that HCQ has known antiviral activity.

The people involved in these antiviral studies were so absolutely blinkered, they couldn t tell apart hydroxychloroquine and hydroxyquinoline, and ended up giving patients toxic doses of HCQ. Not to mention, the very sick patients enrolled for these antiviral studies had sepsis, and hardly any virus left in their bodies. They were well past the point of effectiveness of antivirals, having already been symptomatic for over a week, their viral loads having declined to negligible levels, a disordered immune response causing continuous damage to the lining of their blood vessels.

In 2020, Klaus Schwab and Thierry Malleret published a book called COVID-19: The Great Reset, which was basically a wish list of WEF policies that could be implemented using the virus as a convenient crisis to leapfrog off of.

“COVID-19: The Great Reset” is a guide for anyone who wants to understand how COVID-19 disrupted our social and economic systems, and what changes will be needed to create a more inclusive, resilient and sustainable world going forward. Klaus Schwab, founder and executive Chairman of the World Economic Forum, and Thierry Malleret, founder of the Monthly Barometer, explore what the root causes of these crisis were, and why they lead to a need for a Great Reset.Theirs is a worrying, yet hopeful analysis. COVID-19 has created a great disruptive reset of our global social, economic, and political systems. But the power of human beings lies in being foresighted and having the ingenuity, at least to a certain extent, to take their destiny into their hands and to plan for a better future. This is the purpose of this book: to shake up and to show the deficiencies which were manifest in our global system, even before COVID broke out.”Erudite, thought-provoking and plausible” — Hans van Leeuwen, Australian Financial Review (Australia)”The book looks ahead to what the post-coronavirus world could look like barely four months after the outbreak was first declared a pandemic” — Sam Meredith, CNBC (USA) “The message that the pandemic is not only a crisis of enormous proportions, but that it also provides an opportunity for humanity to reflect on how it can do things differently, is important and merits reflection”– Ricardo Avila, Portafolio (Colombia) “A call for political change in the post-pandemic world”– Ivonne Martinez, La Razon (Mexico)”History has shown, the book argues, that pandemics are a force for radical and lasting change”– Mustafa Alrawi, The National (UAE)

As we rolled into 2021, the madness of vaccine mandates and passes began, with people facing ostracization, joblessness, and restriction of free movement as the price of vaccine refusal.

David E. Martin, the CEO of M-CAM, released a document showing that every part of SARS is a patented product.

Anthony Fauci stood up before Congress and perjured himself multiple times, denying that any GOF research took place at the WIV with NIH funding.

The NIH later admitted that GOF research did take place.

NIH admits US funded gain-of-function in Wuhan despite Fauci s denials

According to Tabak, the NIH had reviewed EcoHealth s research plan in advance of approving the grant but claims it wasn t subjected to additional review at the time as it didn t fit the definition of research involving enhanced pathogens of pandemic potential because these bat coronaviruses had not been shown to infect human.

Tabak said if EcoHealth had alerted NIH to the growth, it would have prompted a review to determine if the research plan should be re-evaluated.

Recently, an anonymous scientist came forward with a claim that SARS-CoV-2 s furin cleavage site contains a 19-nt sequence as its reverse complement, CTCCTCGGCGGGCACGTAG, which aligns with the sequence of a Moderna patented cell line.

Arkmedic’s blog
How to BLAST your way to the truth about the origins of COVID-19
I ve been meaning to write this blog for ever. Well, at least since Prashant Pradhan (a wonderful, honest and brave genomics scientist) raised the possibility back in February 2020 that the SARS-Cov2 virus was man made. And we have seen multiple confirmatory pieces that the virus was made in a lab, one of the better ones here on&

Read more

There is now a paper published in Frontiers in Virology on this matter.

Frontiers in Virology – MSH3 Homology and Potential Recombination Link to SARS-CoV-2 Furin Cleavage Site

Among numerous point mutation differences between the SARS-CoV-2 and the bat RaTG13 coronavirus, only the 12-nucleotide furin cleavage site (FCS) exceeds 3 nucleotides. A BLAST search revealed that a 19 nucleotide portion of the SARS.Cov2 genome encompassing the furing cleavage site is a 100% complementary match to a codon-optimized proprietary sequence that is the reverse complement of the human mutS homolog (MSH3). The reverse complement sequence present in SARS-CoV-2 may occur randomly but other possibilities must be considered. Recombination in an intermediate host is an unlikely explanation. Single stranded RNA viruses such as SARS-CoV-2 utilize negative strand RNA templates in infected cells, which might lead through copy choice recombination with a negative sense SARS-CoV-2 RNA to the integration of the MSH3 negative strand, including the FCS, into the viral genome. In any case, the presence of the 19-nucleotide long RNA sequence including the FCS with 100% identity to the reverse complement of the MSH3 mRNA is highly unusual and requires further investigations.

Andrew Huff, the former Vice President of EcoHealth Alliance, has come forward with a whistleblower complaint stating that Peter Daszak was a CIA asset.

EcoHealth Alliance: Whistleblower Exposes Corruption

In late October 2021, Huff says he came forward as a material witness and whistleblower related to numerous unethical and criminal behaviors that took place at EcoHealth Alliance. EcoHealth Alliance engaged in fraud against the U.S. government (Timecard Fraud and contract reimbursement fraud). Huff brought them to the attention of Peter Daszak, Dr. Aleksei Chamura, and CFO Harvey n. After raising these issues at the meeting, Harvey Kasdan went home from work, had a heart attack, and died.

Twitter avatar for @AGHuffDr. Andrew Huff @AGHuff

Here is a copy of the Whistle Blower complaint that I submitted to Senator Gary Peter’s Office. I would like to testify on this critical matter as soon as reasonably possible.

@SenEdMcBroom @SenGaryPeters @RepJackBergman @LeeSmithDC @SenStabenow @joerogan

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As of late, Moderna s stock price appears to be in rapid decline. StEphane Bancel deleted his Twitter account. Pfizer appear to be griping that they have to publish their data and thus be exposed to further scrutiny.

I, myself, cannot come up with a rational response to any of this without resorting to vigorous, sailor-like profanity that would sear your eyeballs. So, instead, I ll leave it up to the reader to decide what is actually going on here.

What do DARPA want with our brains?

All of this has a much darker undercurrent to it (if that could even be considered possible) when one draws the Lieber connection.

In 2020, a Harvard scientist by the name of Charles Lieber was indicted by the DOJ on charges of making false statements. He had taken money from China s Thousand Talents Plan, against the terms of his DOD grants. Charles Lieber received grants from DARPA, ONR, AFOSR, NIH, and MITRE, but he also double-dipped and took money from the CCP, hence the charges against him.

Harvard University Professor Indicted on False Statement Charges

It is alleged that, unbeknownst to Harvard University, beginning in 2011, Lieber became a Strategic Scientist at Wuhan University of Technology (WUT) in China.  He later became contractual participant in China s Thousand Talents Plan from at least 2012 through 2015.  China s Thousand Talents Plan is one of the most prominent Chinese talent recruitment plans designed to attract, recruit, and cultivate high-level scientific talent in furtherance of China s scientific development, economic prosperity and national security.  According to court documents, these talent recruitment plans seek to lure Chinese overseas talent and foreign experts to bring their knowledge and experience to China, and they often reward individuals for stealing proprietary information.  Under the terms of Lieber s three-year Thousand Talents contract, WUT allegedly paid Lieber a salary of up to $50,000 USD per month, living expenses of up to 1 million Chinese Yuan (approximately $158,000 USD at the time) and awarded him more than $1.5 million to establish a research lab at WUT.  In return, Lieber was obligated to work for WUT not less than nine months a year by declaring international cooperation projects, cultivating young teachers and Ph.D. students, organizing international conference[s], applying for patents and publishing articles in the name of [WUT].  

Charles Lieber worked under a cover story. He claimed to be working on silicon nanowire batteries for the Chinese, but no one can recall him ever working on batteries of any kind.

Why did a Chinese university hire Charles Lieber to do battery research?

In fact, one U.S. nanoscientist and former student of Lieber’s says: “I have never seen Charlie working on batteries or nanowire batteries.” (The scientist asked that their name not be used because of the sensitivity surrounding Lieber’s case.)

Charles Lieber s research actually involves something called bionanotechnology, which is the Frankensteinian blending of living tissue with artificial components (i.e. semiconductors, quantum dots, nanoparticles, synthetic polymers, et cetera), on the molecular level.

Charles Lieber s own papers explicitly describe the potential for his silicon nanowires to be used as biosensors, or even as brain-computer interfaces, (also known as brain-machine interfaces, or, in cyberpunk science fiction parlance, neural laces ):

Nanowire probes could drive high-resolution brain-machine interfaces

Brain-machine interfaces (BMIs) can serve as bidirectional connections that output electrical signals of brain activity or input electrical stimuli to modulate brain activity in concert with external machines, including computer processors and prosthetics, for human enhancement[1,2]. Reading electrical activity from neurons is the foundation of many BMI applications, such as brain mapping, that aims to understand brain functions by decoding the communication between neurons. Reading and processing this activity is also key to neural prosthetics in which brain activity is used to control devices such as artificial limbs. For these BMI applications, most of the in vivo recording tools used today read extracellular neural activity by detecting suprathreshold action potential signals that leak outside of neurons (Fig. 1a (i)), while critical subthreshold events, such as synaptic potentials and dendritic integration, remain hidden [3]. To achieve the most information-rich readings, which could provide more detailed mapping of brain function and the finest control of neural prosthetics, electronic devices need to provide access to intracellular signals from multiple neurons comprising the neuronal circuits and networks of the brain [4].

How is this relevant to COVID-19?

Charles Lieber is a colleague of Robert Langer, one of the co-founders of Moderna. The two of them are pictured here, left and right, with Daniel Kohane in the center.

These three worked on a paper together to create cybernetic heart tissue scaffolds with biosensor functionality.

Cyborg Tissue Monitors Cells

The cyborg-like tissue, described online at Nature Materials, supports cell growth while simultaneously monitoring the activities of those cells. It could improve in vitro drug screening by allowing researchers to track how cells in a three-dimensional environment respond to drugs in real time, the authors say. It may also be a first step toward prosthetics that communicate directly with the nervous system, and tissue implants that sense and respond to injury or disease.

Elon Musk s Neuralink presentation made waves when people started to consider the human applications, but even Neuralink is as barbaric as trephination next to the military s craniotomy-free, wireless nanoparticle BCI research.

Enter DARPA s BRAIN Initiative, and the N3 (Next-Generation Nonsurgical Neurotechnology) program.

Next-Generation Nonsurgical Neurotechnology

Whereas the most effective, state-of-the-art neural interfaces require surgery to implant electrodes into the brain, N3 technology would not require surgery and would be man-portable, thus making the technology accessible to a far wider population of potential users. Noninvasive neurotechnologies such as the electroencephalogram and transcranial direct current stimulation already exist, but do not offer the precision, signal resolution, and portability required for advanced applications by people working in real-world settings. The envisioned N3 technology breaks through the limitations of existing technology by delivering an integrated device that does not require surgical implantation, but has the precision to read from and write to 16 independent channels within a 16mm3 volume of neural tissue within 50ms. Each channel is capable of specifically interacting with sub-millimeter regions of the brain with a spatial and temporal specificity that rivals existing invasive approaches. Individual devices can be combined to provide the ability to interface to multiple points in the brain at once.

The grant proposal for N3 is very informative on the nature of the technology desired by DARPA:

Broad Agency Announcement Next-Generation Non-Surgical Neurotechnology (N3) BIOLOGICAL TECHNOLOGIES OFFICE HR001118S0029

TA2 involves the development of a system that includes a nanotransducer placed on or near neurons of interest and an integrated sensor/stimulator device that sits outside the skin. The nanotransducer may include technologies such as, but not limited to, self-assembled/molecular/biomolecular/chemical nanoparticles, or viral vectors. These nanotransducers must be delivered in a minutely invasive (nonsurgical) manner, which may include ingestion, injection, or nasal administration, and involve technology that includes self-assembly inside the body. While the major TA2 goals of developing neural read out and write in capabilities are similar to the goals from TA1, creating a nanotransducer with an optimal delivery route to the brain is a major additional component. Another major component of TA2 is achieving cell-type specificity.

There are six teams involved in the N3 program; Battelle, Carnegie Mellon University, Johns Hopkins University s Applied Physics Laboratory, PARC, Rice University, and Teledyne.

How close are they to achieving their goal?

Magnetism Plays Key Roles in DARPA Research to Develop Brain-Machine Interface without Surgery

DARPA is preparing for a future in which a combination of unmanned systems, artificial intelligence, and cyber operations may cause conflicts to play out on timelines that are too short for humans to effectively manage with current technology alone, said Al Emondi, the N3 program manager. By creating a more accessible brain-machine interface that doesn t require surgery to use, DARPA could deliver tools that allow mission commanders to remain meaningfully involved in dynamic operations that unfold at rapid speed.  

Nature – Wireless and battery-free technologies for neuroengineering

Tethered and battery-powered devices that interface with neural tissues can restrict natural motions and prevent social interactions in animal models, thereby limiting the utility of these devices in behavioural neuroscience research. In this Review Article, we discuss recent progress in the development of miniaturized and ultralightweight devices as neuroengineering platforms that are wireless, battery-free and fully implantable, with capabilities that match or exceed those of wired or battery-powered alternatives. Such classes of advanced neural interfaces with optical, electrical or fluidic functionality can also combine recording and stimulation modalities for closed-loop applications in basic studies or in the practical treatment of abnormal physiological processes.

Very close.

Incidentally, the head of Battelle s N3 team, Gaurav Sharma, was also involved in DTRA s Blood-Brain Barrier Program. Yes, the very same Defense Threat Reduction Agency that funded EcoHealth Alliance.

What did that program seek to accomplish?

Early Successes of DTRA s Blood-Brain Barrier Program Suggest New Countermeasures

The program aims to understand the effects of nerve agents and alphaviruses on the blood-brain barrier and find new transport pathways to deliver appropriate therapeutics into the CNS. The early successes of JSTO s program allows researchers to better assess the risks of emerging threats while enhancing their ability to protect and treat warfighters from a broad range of chemical and biological threats.

That s interesting. What does SARS-CoV-2 Spike, the supposed constituent of the vaccine, do to people s blood-brain barrier?

Elsevier – The SARS-CoV-2 spike protein alters barrier function in 2D static and 3D microfluidic in-vitro models of the human blood brain barrier

Key to our findings is the demonstration that S1 promotes loss of barrier integrity in an advanced 3D microfluidic model of the human BBB, a platform that more closely resembles the physiological conditions at this CNS interface. Evidence provided suggests that the SARS-CoV-2 spike proteins trigger a pro-inflammatory response on brain endothelial cells that may contribute to an altered state of BBB function.

Huh. The SARS-CoV-2 Spike S1 subunit is capable of permeabilizing the blood-brain barrier, opening it up for substances that might want to cross from the bloodstream to the brain.

So, how does all this wireless neural lace business work? It s actually both very complex and surprisingly simple. Nanoparticles that are sensitive to RF, electromagnetism, ultrasound, and/or light are introduced into brain cells, and then, an external encoder/decoder stimulates them wirelessly and reads back the response. It is very much like turning the human brain into a Wacom pen. You know how a Wacom tablet works, right? The tablet generates a field that the pen converts into an electric current by wireless induction. That s why the pens don t need batteries, and it s why nanotransducers implanted in the brain don t need them, either.

Shortly after the Spartacus Letter was posted on ZeroHedge, this absolutely ridiculous CNBC video was published on YouTube:

How small are nanotransducers, actually?

Well, here s one of Battelle s Magnetoelectric Nanotransducers, as seen under an electron microscope:

It s about 20 nanometers across, much smaller than the 120 nanometer diameter of a SARS-CoV-2 virion, and comparable to transistor gate sizes.

To put this in perspective, the inside diameter of a vaccine needle of average gauge is about 0.26 millimeters. That s 260,000 nanometers.

13,000 Magnetoelectric Nanotransducers could fit through a vaccine needle side-by-side.

You see, BCIs capable of two-way communication with a human brain don t just give you superpowers, or let you control drones with your mind, or listen to Katy Perry without headphones or speakers or whatever. They can also be used to manipulate mood, and for social control.

They could also be used to take away people s agency and turn them into, essentially, biological robots, utterly obedient to government and open to any manner of sadistic abuse.

This is not an exaggeration. It is a fact.

The ethics of brain computer interfaces

Reports have surfaced about a minority of people who undergo DBS for Parkinson s disease becoming hypersexual, or developing other impulse-control issues. One person with chronic pain became deeply apathetic after DBS treatment. DBS is very effective, Gilbert says, to the point that it can distort patients perceptions of themselves. Some people who received DBS for depression or obsessive compulsive disorder reported that their sense of agency had become confused2. You just wonder how much is you anymore, said one. How much of it is my thought pattern? How would I deal with this if I didn t have the stimulation system? You kind of feel artificial.

Neuroethicists began to note the complex nature of the therapy s side effects. Some effects that might be described as personality changes are more problematic than others, says Maslen. A crucial question is whether the person who is undergoing stimulation can reflect on how they have changed. Gilbert, for instance, describes a DBS patient who started to gamble compulsively, blowing his family s savings and seeming not to care. He could only understand how problematic his behaviour was when the stimulation was turned off.

Profound behavioral changes have already been observed in patients with DBS electrodes implanted in their heads. How far could they go if they had access to fine-grained stimulation of individual clusters of neurons?

A bioethicist by the name of Dr. James Giordano (who is well-acquainted with DARPA s research) is very concerned about all of this. That s why he gives horrifying lectures in front of West Point cadets like this one:

Are they already wargaming the military applications of weaponized neuroS/T, investigating the possibility of using nanoparticles to attack rival powers by making their own citizens go berserk? Yes, of course they are.

COGNITIVE WARFARE – June-November 2020 FranAois du Cluzel

The use of neuroS/T for military and intelligence purposes is realistic, and represents a clear and present concern. In 2014, a US report asserted that neuroscience and technology had matured considerably and were being increasingly considered, and in some cases evaluated for operational use in security, intelligence, and defense operations. More broadly, the iterative recognition of the viability of neuroscience and technology in these agenda reflects the pace and breadth of developments in the field. Although a number of nations have pursued, and are currently pursuing neuroscientific research and development for military purposes, perhaps the most proactive efforts in this regard have been conducted by the United States Department of Defense; with most notable and rapidly maturing research and development conducted by the Defense Advanced Research Projects Agency (DARPA) and IntelligenceAdvanced Research Projects Activity (IARPA). To be sure, many DARPA projects are explicitly directed toward advancing neuropsychiatric treatments and interventions that will improve both military and civilian medicine. Yet, it is important to note the prominent ongoing and expanding efforts in this domain by NATO European and trans-Pacific strategic competitor nations.

&

A cognitive attack is not a threat that can be countered in the air, on land, at sea, in cyberspace, or in space. Rather, it may well be happening in any or all of these domains, for one simple reason: humans are the contested domain.

Naturally, this sort of escalation of neuroscience to the level of a new arms race will create all kinds of insane justifications in the minds of policymakers and intelligence officials. Populist unrest can be recast as inauthentic, the result of undetectable enemy action. Dissidents can be rounded up and injected with mind-control nanoparticles to make them friendly to the state again, and to cure whatever the Russian or Chinese nanoparticles did to them to make them get so upset and wave signs in the street in the first place.

I ask you, the reader, is this a world you want to live in? One where governments see your own body, your own mind, as not off-limits for direct tampering to achieve military and political objectives? A world where maniacs describe your own body, your own flesh and blood, not as something sacrosanct, but as little more than the contested domain of a new type of warfare that you never even knew existed?

No. We say no.

If governments are crossing this line, then they have become the enemies of all mankind.

-Spartacus

 

 

 

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Jan 112022
 
 January 11, 2022  Posted by at 10:00 am Finance Tagged with: , , , , , , ,  84 Responses »


Rembrandt van Rijn The Adoration of the Magi 16xx

 

USCF Covid Doctor: The Hospital Surge Isn’t What You May Think (SFGate)
Gloomsters Admit They Were Wildly Wrong About 75,000 Omicron Deaths (DM)
Huge Number of Vax Deaths & It’s Getting Worse – Dr. Pierre Kory (USAW)
Numbers Killed by Vaccines Much Worse than We Thought (Bhakdi, Yeadon)
Double Vaccinated Have Double the Infection Rate (DS)
Omicron 105% More Transmissible Than Delta, Says Study (BS)
Has The Great Barrington Declaration Been Vindicated? (Unherd)
T-Cells From Common Colds Can Provide Protection Against Covid-19 (R.)
Health Officials Let Covid-infected Staff Stay On The Job (AP)
Light It Up! (Kunstler)
Where’s The Wood? (Denninger)
80% of Airline Pilots Aren’t Going To Take The Booster (Kirsch)
Military Documents About Gain Of Function Contradict Fauci Testimony (Veritas)
International Finance Leaders Hold ‘War Game’ Exercise (CHD)

 

 

Trends: 99% of Omicron infections are harmless. Vaccine injuries and deaths are now coming to the fore. Can the narrative be controlled for much longer?

 

 

“Omicron has learned how to read!!! Of the 30 mutations in its spike gene, 24 have been characterized in scientific publications. Also, it borrowed defining mutations from alpha, beta, delta, eta, epsilon, iota and mu. Even though it was in hiding when these variants emerged.”

 

 

 

Pfizer CEO Vaccine 1.1

 

 

The greatest proponents for all of these ruinous Covid measures… will be rewriting their own history..
https://twitter.com/i/status/1480295361549946880

 

 

Rewriting as we speak….
https://twitter.com/i/status/1480682158238752770

 

 

 

 

“As currently reported, COVID hospitalization rates greatly exaggerate COVID burden. Incidental positives account for large majority of hospitalized cases in both LA and Bay Area.”

“The vast majority of COVID-plus patients I take care of need no medical care and are quickly discharged home with reassurance.”

USCF Covid Doctor: The Hospital Surge Isn’t What You May Think (SFGate)

On Saturday, in response to hospitals begging for relief from a massive staffing crisis, the California Department of Public Health announced that most hospitals and skilled nursing facilities can bring COVID-positive and exposed staff back to work without testing or quarantines. The staffers must be asymptomatic, are required to wear N95 masks and are encouraged to work with patients who are already COVID-positive as much as possible. This news might come as a surprise to people who have been reading dire warnings about omicron and some public health officials’ pleas to cancel plans and stay home. Many public health officials have argued these measures are necessary to prevent hospitals from being overwhelmed with COVID patients.

Indeed, for the past few weeks, San Francisco hospitals have been in dire straits. But it’s not because people are sick — it’s because of staffing shortages driven by overly strict state quarantine rules, the director of COVID response at UCSF’s emergency department said. After reviewing the charts of every COVID-positive patient at UCSF hospitals on Jan. 4, Dr. Jeanne Noble, an associate professor of emergency medicine at UCSF, determined that 70% of them were in the hospital for other reasons. “The real COVID crisis that our hospitals are facing is a severe staffing shortage that is compromising the quality of our care,” Noble said Friday, shortly before the policy change was announced. Staffing shortages are so severe that California is considering canceling elective surgeries, as happened during the worst of last year’s peak.

“The crisis from the Omicron peak is not generated by serious COVID illness in regions with highly vaxxed populations,” Noble wrote in an email to SFGATE. “The crisis we are suffering in the Bay Area is largely driven by disruptive COVID policies that encourage asymptomatic testing and subsequent quarantines. … The vast majority of COVID-plus patients I take care of need no medical care and are quickly discharged home with reassurance.” It’s true that case counts are shattering records set last year, and Noble predicts the peak is still a week away. But fewer people are hospitalized with COVID today in California compared with this time last year. And, especially in highly vaccinated areas, few of those patients are actually in the hospital because of COVID illness. In LA, where 71% of eligible people are fully vaccinated, two-thirds of hospital cases were caught on screening for the virus, the LA Times reported.

[..] she identified 44 hospitalized patients (both adults and children) with COVID. Of those, just 13 were admitted because of COVID. “I do not expect that number to increase substantially, or become unmanageable in the coming week,” she wrote. “The death rate in California is actually falling. And the predicted peak of cases is only about a week away.” The remaining 31, or 70%, of patients tested positive after being admitted for unrelated reasons, including a hip fracture and a bowel obstruction. They’re all “completely asymptomatic or minimally symptomatic,” Noble said. “[Emergency departments] are flooded with the worried well that are simply seeking testing and reassurance,” she added. “I have not intubated a single COVID patient during this Omicron surge. We have a total of 5 patients with COVID on ventilators across our 4 hospitals. An average of 1.25 intubated COVID patients per hospital is a good news story.”

Read more …

Neil Ferguson, anyone?

Gloomsters Admit They Were Wildly Wrong About 75,000 Omicron Deaths (DM)

The scientists who warned that Britain had little option but to impose severe restrictions or face tens of thousands of deaths from Omicron were last night in retreat. First, modellers who advise the Government said winter deaths from the highly transmissible variant would be ‘substantially’ lower than they had originally believed, then Independent SAGE, a group of Left-leaning scientists who have pushed for lockdowns, distanced themselves from the need to impose further curbs. Before Christmas, epidemiologists at the London School of Hygiene and Tropical Medicine produced a series of dire scenarios in which they warned Omicron could lead to between 25,000 and 75,000 deaths by the end of April. But one of its leading modellers said last night he believes the true figure will be far lower, mainly due to Omicron being less lethal than originally feared.

Dr Davies said that since mid-December he and his team had been in constant communication with senior civil servants and government scientific advisers, discussing emerging data that pointed towards Omicron having lower severity than originally feared, and the implications this could have for policy Tory MP William Wragg, a member of the party’s Covid Recovery Group, said the U-turn provided evidence that many in the scientific community had been too gloomy about the threat from coronavirus. ‘Once again, it appears that certain scientists and experts so quick to spread gloom and panic at the arrival of Omicron are having to come to terms with a reality that is far from the catastrophe they were predicting,’ he said. ‘It all shows that Boris Johnson and his Cabinet were right to avoid condemning us to another lockdown with the dismal effects on people’s livelihoods and liberties.’

The School of Hygiene’s team built its original models – published on December 11 – on the assumption that Omicron was as naturally lethal as the Delta strain, meaning it would kill the same proportion of unvaccinated people who had not been exposed to Covid before. Dr Davies argued that while South African doctors were already finding Omicron appeared to be less severe, the reports were ‘anecdotal’ so the School of Hygiene’s supposition was ‘a reasonable assumption to make at the time’. Over the past month, however, considerable evidence has built up that Omicron is less dangerous. This includes statistical studies by Imperial College London, Edinburgh University and the UK Health Security Agency, as well as research from South Africa and Denmark. Laboratory studies have also found Omicron is less adept at infecting the lungs.

Read more …

This is the story coming out, that needs to be suppressed.

Huge Number of Vax Deaths & It’s Getting Worse – Dr. Pierre Kory (USAW)

Former Pfizer VP Dr. Michael Yeadon said this week, “Max vaccination is leading to mass death.” Dr. Kory agrees and explains, “It’s not only data from a life insurance company that came out this week that is based on CDC data that can’t be explained by Covid alone, there are huge increases of dying in this country this year. . . . They have done huge analysis of the European mortality data as well as the U.S. mortality data and they controlled for vaccination status. They found that for every age range that they looked at, the all-cause mortality of the vaccinated were increased over the unvaccinated. All-cause mortality and that means that you are more likely to die of something if you are vaccinated. . . .


All-cause mortality are coming out of actual databases by credible scientists. You have life insurance companies showing the data, and you have our own federal government showing unexplained large rises in dying. . . . Don’t you think a good scientific question and a good hypothesis to test would be ‘Could these be the vaccines?’ The answer is ‘the vaccines,’ and I cannot find a better fit to answering that hypothesis than that, it’s this mass explosion of this vaccination policy with single, double and booster shots. It’s going like wildfire through the population. If the mortality of the vaccinated is higher than the unvaccinated, you have the data that you can safely and confidently conclude the vaccines are associated with and causing death.”

Read more …

But it will be hard to keep it silent much longer.

Numbers Killed by Vaccines Much Worse than We Thought (Bhakdi, Yeadon)

My good friend Dr Sucharit Bhakdi, with whom we and others wrote a series of open letters to the European Medicines Agency, is utterly distraught. Listen carefully. He and his colleague, a pathologist, have confirmed that, even in people who’ve died post-covid-19 vaccination and where their death was not attributed to the adverse effects of vaccination, in almost all cases they DID die as a result of the vaccination. The numbers killed by these vaccines is much worse than what we thought, already. But it’s what they’ve just discovered that’s much worse. We knew of blood clots from expressing spike protein. We were aware of autoimmune attacks on ones own tissues expressing spike protein to which our killer lymphocytes were primed, such as myocarditis.

But what’s new is the revelation that lymph node cells are also being invaded by the gene-based agents and marking THEM for auto destruction. When you destroy that part of the immune system, which we loosely call “immune surveillance”, every manner of nasty, latent infections, by viruses & also bacteria, explode, uncontrolled. Hundreds of millions of people are going to die of unrestrained tuberculosis, Epstein Barr virus, toxoplasmosis etc etc etc AND on top of this, the daily accidental production of cancer cells, normally deleted swiftly by immune surveillance, before they can divide, ceases. Guess what happens next? I don’t care where you’ve sat during this ridiculous “pandemic”. Whether you’ve gone along with it, knowing it was an overreaction. Or even in ignorance.

I am telling you right now: IF YOU PERSONALLY HAVING WATCHED THIS CHOOSE TO SETTLE BACK TO WATCH A FILM, INSTEAD OF CALLING SOME PEOPLE YOU KNOW & TELLING THEM ABOUT IT, THE END OF HUMANITY IS A SHARED BURDEN WITH THE PERPETRATORS. Please put this on every platform. Swamp the ‘fact checkers’. Please do it now. Rescue our civilization while there are innocents to save, ESPECIALLY our children and grandchildren. Thank you sincerely,

Read more …

And triple?

Double Vaccinated Have Double the Infection Rate (DS)

The double-vaccinated are almost twice as likely to be infected as unvaccinated people, data from Iceland shows. This is the same pattern as found in data from the U.K. Thorsteinn Siglaugsson has written about the trend in Morgunbladid, the main national newspaper in Iceland, and put up a translation on his website.

“After December 20th, the 14-day incidence of COVID-19 infection by vaccination status took a very unexpected turn in Iceland. The infection rate per 100,000 of fully vaccinated adults with booster is now eleven times higher than on December 20th, and the infection rate of double-vaccinated adults seven times higher. At the same time, infections among unvaccinated people have grown by a factor of 2.6 only. Among children, we see a similar change: a tenfold increase among the fully vaccinated while the rate among the unvaccinated is 2.4 times higher than on December 20th. This change can hardly be explained away by changes in behaviour, such a sudden and decisive change of behavior between groups is impossible. It is also unlikely that testing has suddenly increased this sharply among some groups and not others.

We know the protection against infection from vaccination wanes rapidly, but it is out of the question that it should drop so suddenly. The most likely explanation is the new Omicron variant. Foreign data also indicate that the currently available vaccines have little or no effect against Omicron infection. The data published on covid.is are weighted; the different size of the groups is adjusted for. This means we can use them to conclude regarding probability of infection. At present, triple-vaccinated people are only 30% less likely to get infected than unvaccinated adults, and for vaccinated children the difference is only 15%. This small difference decreases rapidly in both groups. The biggest news, however, is that double-vaccinated people are now 90% more likely to get infected than the unvaccinated. This suggests that the protection provided by two doses of vaccine is in fact less than none, it is the opposite.”

After Siglaugsson’s article was published on January 8th, the Icelandic Chief Epidemiologist claimed the finding was an artefact of the official data overestimating the number of unvaccinated people. However, Siglaugsson suggests the overestimate would have to be an implausible 90% to bring the infection rates level.

Read more …

Double Vaccinated =2x. Omicron=2x. Are we looking at 22?

Omicron 105% More Transmissible Than Delta, Says Study (BS)

The Omicron Covid-19 variant may be 105 per cent more transmissible than Delta, according to a research by French scientists. The study, published on the medRxiv site and yet to be peer-reviewed, analysed 131,478 tests in France from October 25 to December 18, 2021. The team applied statistical models to variant-specific screening tests and full genome sequencing. They compared the number of infections with the Omicron, Alpha and the Delta variants over a 21-day period. The difference in rate of transmissibility in people with the Delta and Omicron was approximately 105 per cent.


“We estimate that the transmission advantage of the Omicron variant over the Delta variant is more than 105 per cent,” said Samuel Alizon, from Centre for Interdisciplinary Research in Biology (CIRB) France. Further, the results showed that tests consistent with the presence of the Omicron variant exhibit significantly higher cycle threshold Ct values, which could indicate lower amounts of virus genetic material. “Epidemiological modelling indicates that even if the virulence of the Omicron variant is reduced compared to that of the Delta variant, the increase in reproduction number we estimate from the data can has the potential to maintain critical Covid-19 activity at a high level in French hospitals, if not overloading them,” Alizon said adding that “swift mitigation of the epidemic wave” is essential.

Read more …

Not as long as Fauci is around.

Has The Great Barrington Declaration Been Vindicated? (Unherd)

Over the course of the past two years, Italy has implemented some of the strictest and longest lockdowns in the world (indeed, it is the country that “invented” the concept of national lockdown), topping every other Western country in terms of average stringency of anti-Covid measures. Yet Italy is also one of the countries with the highest mortality rate per capita — well above the United Kingdom, Spain, France, Germany, Sweden and several other countries that adopted much less restrictive measures. And there’s evidence that this isn’t despite the lockdowns but, most likely, because of them.

As Piero Stanig and Gianmarco Daniele, two professors at Bocconi University, explain in their book Fallimento lockdown (“Lockdown Failure”), the worst possible thing you can do when dealing with a highly infectious disease that spreads almost exclusively indoors and targets the elderly is to lock old people up inside their homes with other family members, and ban citizens from spending time in arguably the safest place of all: outdoors. In other words, even from the narrow perspective of saving lives, not only were lockdowns not in the collective interest of society, they weren’t even in the interest of those whose lives were actually at risk. Such an outcome was easily predictable. Indeed, the WHO’s 2019 report on pandemic preparedness states that the quarantine of exposed individuals — let alone of the entire population — “is not recommended because there is no obvious rationale for this measure”.

The grotesquery of the global responses becomes even more apparent when we take into account the fact that while governments went out of their way to keep healthy people locked in, chasing runners down solitary beaches or checking shopping trolleys to make sure people were only buying essentials, they all but abandoned those most vulnerable: nursing home residents. According to a recent Collateral Global study, Covid deaths in nursing homes amount on average to a staggering 40% of all Covid deaths in Western countries, despite representing less than 1% of the population. In some countries (Belgium, France, the Netherlands, Slovenia, Spain, Sweden, the UK and the US), more than 5% of all care home residents were killed.

In view of this, it seems obvious that the focused protection approach championed by the Great Barrington Declaration (GBD) — based on “allow[ing] those who are at minimal risk of death to live their lives normally to build up immunity to the virus through natural infection, while better protecting those who are at highest risk” — was the right course of action. It would have avoided inflicting needless pain on workers, women and children through repeated lockdowns, while arguably saving countless lives, by focusing first and foremost on the elderly and especially on nursing homes.

Read more …

Exactly what Great Barrington Professor @SunetraGupta said from the very beginning. The common cold had given many of us some protection against Covid.

T-Cells From Common Colds Can Provide Protection Against Covid-19 (R.)

High levels of T-cells from common cold coronaviruses can provide protection against Covid-19, an Imperial College London study published on Monday has found, which could inform approaches for second-generation vaccines. Immunity against Covid-19 is a complex picture, and while there is evidence of waning antibody levels six months after vaccination, T-cells are also believed to play a vital role in providing protection. The study, which began in September 2020, looked at levels of cross-reactive T-cells generated by previous common colds in 52 household contacts of positive Covid-19 cases shortly after exposure, to see if they went on to develop infection.

It found that the 26 who did not develop infection had significantly higher levels of those T-cells than people who did get infected. Imperial did not say how long protection from the T-cells would last. “We found that high levels of pre-existing T cells, created by the body when infected with other human coronaviruses like the common cold, can protect against Covid-19 infection,” study author Dr Rhia Kundu said. The authors of the study, published in Nature Communications, said that the internal proteins of the SARS-CoV-2 virus which are targeted by the T-cells could offer an alternative target for vaccine makers.

Current Covid-19 vaccines target the spike protein, which mutates regularly, creating variants such as Omicron which lessen the efficacy of vaccines against symptomatic infection. “In contrast, the internal proteins targeted by the protective T-cells we identified mutate much less,” Professor Ajit Lalvani, co-author of the study, said. “Consequently, they are highly conserved between the various SARS-CoV-2 variants, including Omicron. New vaccines that include these conserved, internal proteins would therefore induce broadly protective T cell responses that should protect against current and future SARS-CoV-2 variants.”

Read more …

But only when vaccinated… You know, to replace the healthy unvaxxed…

Health Officials Let Covid-infected Staff Stay On The Job (AP)

Health authorities around the U.S. are increasingly taking the extraordinary step of allowing nurses and other workers infected with the coronavirus to stay on the job if they have mild symptoms or none at all. The move is a reaction to the severe hospital staffing shortages and crushing caseloads that the omicron variant is causing. California health authorities announced over the weekend that hospital staff members who test positive but are symptom-free can continue working. Some hospitals in Rhode Island and Arizona have likewise told employees they can stay on the job if they have no symptoms or just mild ones. The highly contagious omicron variant has sent new cases of COVID-19 exploding to over 700,000 a day in the U.S. on average, obliterating the record set a year ago.

The number of Americans in the hospital with the virus is running at about 110,000, just short of the peak of 124,000 last January. Many hospitals are not only swamped with cases but severely shorthanded because of so many employees out with COVID-19. At the same time, omicron appears to be causing milder illness than the delta variant. Last month, the Centers for Disease Control and Prevention said that health care workers who have no symptoms can return to work after seven days with a negative test, but that the isolation time can be cut further if there are staffing shortages. France last week announced it is allowing health care workers with mild or no symptoms to keep treating patients rather than isolate.

[..] In California, the Department of Public Health said the new policy was prompted by “critical staffing shortages.” It asked hospitals to make every attempt to fill openings by bringing in employees from outside staffing agencies. Also, infected workers will be required to wear extra-protective N95 masks and should be assigned to treat other COVID-19-positive patients, the department said. “We did not ask for this guidance, and we don’t have any information on whether hospitals will adopt this approach or not,” said Jan Emerson-Shea, a spokesperson for the California Hospital Association. “But what we do know is that hospitals are expecting many more patients in the coming days than they’re going to be able to care for with the current resources.”

Cali
https://twitter.com/i/status/1480547659895169038

Read more …

“The ballyhooed and mandated vaxxes apparently have the ability to kill and maim people who have taken them long after Covid-19 exits stage-left.”

Light It Up! (Kunstler)

We are at a strange pass in The Saga of Covid. It seems the spikey virus wants to leave center stage… is weary of all the attention… wants to fade into the eternal parade of microorganisms that cozily coexist within the human life-stream — like Tony Fauci’s HIV, a fellow traveler in the old-time throng of human viruses, now semi-retired, and yet still every bit as mysterious in the actual mechanism of AIDS as it was when Dr. Fauci pinned his NIAID distinguished service medal on its elusive bosom, so to speak (but you’d have to read Bobby Kennedy’s book on Fauci to get the drift of that). Omicron is sweeping the country, as love once did in George Gershwin’s day. (We are a different country now, as anyone tuned into the Turner Classics Movie channel can discover.)

Omicron: the 36-hour head cold that Covid-19 has been demoted to. Omicron: a mere wise-cracking gecko compared to the roaring dragon that was Covid-19 in the winter of 2020. Omicron: kind of an embarrassment to “vaccine” tyrants who still seek to jab every arm on earth, and at ever-shortening intervals — like a med school version of The Sorcerer’s Apprentice, only with syringes running amok instead of brooms. The Party-of-Chaos (the one headed by the ectoplasmic “Joe Biden”) does not want to let go of Covid-19, its Swiss army knife of destruction. With Covid-19, you can push people around and mess with their lives every which way, shut down their businesses, lock them in their homes, screw them out of their livelihoods, delete their reputations, board-up their social venues, cancel their careers, revoke their licenses, drag them into court, fine them into penury, cram them into prison camps, and much more.

[..] Which brings us back to the virus. In The Saga of Covid there is a monster under the bed. The ballyhooed and mandated vaxxes apparently have the ability to kill and maim people who have taken them long after Covid-19 exits stage-left. We don’t really know how this works out, but we have plenty of clues: kids dropping dead of heart attacks, pro athletes, ditto, the VAERS numbers reporting over 21,000 vaccine-implicated deaths (out of a grossly under-reported actual figure) plus over a million adverse reaction reports (ditto under-reported). The time may not be far off when we make the ghastly discovery that the “vaccines” actually killed more Americans than the virus did.

Read more …

“There were safety signals — screaming red flags — in September of 2020, four months before the first shots went into arms on the EUAs and during the trials themselves.”

Where’s The Wood? (Denninger)

You know, wood, nails, saws, hammers…. and boiled rope? Now Pfizer and Moderna’s CEOs are apparently on Corrupt National Bull**** crowing that a fourth shot is necessary. May I remind you of the lies? How did all this work out for AOC? Or Geraldo, both of whom were “true believers” that if you got the first two shots you absolutely would not get *****. Both got ***** recently after taking the booster, which was the “second” set of lies. There are many words you may choose to describe a drug you have taken three times within a year and shortly thereafter get the disease but if you use the word “*******” given that personal record of ineffectiveness you deserve an immediate Clue-by-4 to the face in a (likely futile) attempt to correct your vapid stupidity.

Nobody in their right mind would expect a condom that only lasts for 15 seconds of sex before breaking to prevent pregnancy or deter transmission of STDs. That’s how ****ing stupid everyone is being at this point with regard to the jabs. I remind you that the original EUA studies were not designed to, and thus did not, demonstrate that the jabs were sterilizing — that is, that having taken them you would neither get or transmit *****-19. The representations that they in fact had that property were knowing, intentional lies; there was no evidence that was the case and in fact the trials were never intended to show that. Even worse was that although the entire case for the EUAs was built around reducing the risk of severe disease the sub-group of people who need that risk reduction — the seriously morbid — were deliberately excluded from the trials! We thus had zero evidence as to whether what we did observe in healthy young adults would translate into older, fatter, sicker people.

The entire edifice built around the jabs was in fact born of lies. Lies repeated by Donald Trump, Geraldo, AOC, Joe Biden, Anthony Fauci and thousands of others. They were not errors or mistakes; they were lies. In addition to the lies on efficacy the manufacturers and so-called “experts” have also lied about safety. There were safety signals in the original trials but they were deliberately ignored. There were safety signals — screaming red flags — in September of 2020, four months before the first shots went into arms on the EUAs and during the trials themselves. Said science strongly implicated the spike protein alone being pathogenic and that in the circulation it was specifically harmful to the endothelial layer that is in all blood vessels. It was known in December of 2020 that the presence of the spike protein in the circulation could lead the body to attack its own circulatory system. We knew at the same time that viremia, that is, virus in the bloodstream, was essentially never happening except in severe, critical and fatal *****-19 infections and that when it killed you this was usually the means by which it happened.

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” If 20% of the pilots walk off the job, it’s the end of the commercial airline industry.”

80% of Airline Pilots Aren’t Going To Take The Booster (Kirsch)

I interviewed Latane Campbell who is a pilot for a major US airline. Key points:

  • He knows about 100 pilots and 80% are not going to take the booster. They’ll quit if they are forced to take it.
  • Virtually all of the pilots know that masks are completely useless.
  • The pilots all take off their masks as soon as they close the cockpit door. Wearing masks makes flying dangerous.
  • If 20% of the pilots walk off the job, it’s the end of the commercial airline industry.
  • The airlines tried to strong arm the pilots into taking the vaccines or else. The pilots resisted and the airlines immediately recanted allowing religious and medical exemptions.
  • The more you vaccinate, the sicker people get. Most pilots have figured this out.

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DARPA.

Military Documents About Gain Of Function Contradict Fauci Testimony (Veritas)

Project Veritas has obtained startling never-before-seen documents regarding the origins of COVID-19, gain of function research, vaccines, potential treatments which have been suppressed, and the government’s effort to conceal all of this. The documents in question stem from a report at the Defense Advanced Research Projects Agency, better known as DARPA, which were hidden in a top-secret shared drive. DARPA is an agency under the U.S. Department of Defense in charge of facilitating research in technology with potential military applications. Project Veritas has obtained a separate report to the Inspector General of the Department of Defense written by U.S. Marine Corp Major, Joseph Murphy, a former DARPA Fellow.

The report states that EcoHealth Alliance approached DARPA in March 2018, seeking funding to conduct gain of function research of bat borne coronaviruses. The proposal, named Project Defuse, was rejected by DARPA over safety concerns and the notion that it violates the basis gain of function research moratorium. According to the documents, NIAID, under the direction of Dr. Fauci, went ahead with the research in Wuhan, China and at several sites across the U.S.

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IMF,BIS,WEF.

International Finance Leaders Hold ‘War Game’ Exercise (CHD)

High-level international banking officials and organizations last month gathered in Israel for a global “war game” exercise simulating the collapse of the global financial system. The tabletop exercise was reminiscent of “Event 201” — the pandemic simulation exercise that took place in October 2019, shortly before COVID-19 entered the global scene. The “Collective Strength” initiative was held for 10 days, beginning Dec. 9, 2021, at the Israeli Finance Ministry in Jerusalem. It was relocated to Jerusalem from the Dubai World Expo over concerns about the Omicron variant. Israel led a 10-country contingent that also included treasury officials from the U.S., Austria, Germany, Italy, the Netherlands, Switzerland, Thailand and the United Arab Emirates.

Representatives from supranational organizations, such as the International Monetary Fund (IMF), World Bank and Bank of International Settlements (BIS), also participated. Described as a simulated “war game,” the exercise sought to model the response to various hypothetical large-scale cyberattacks on the global financial system, including the leaking of sensitive financial data on the “Dark Web,” hacks targeting the global foreign exchange system, and subsequent bank runs and market chaos fueled by “fake news.” However, the main theme of “Collective Strength” appears not so much the simulation of such cyberattacks but, as the name of the initiative implies, the strengthening of global cooperation in cybersecurity and the financial sector.

As reported by Reuters, participants in the simulation discussed multilateral responses to a hypothetical global financial crisis. Proposed policy solutions included debt repayment grace periods, SWAP/REPO agreements, coordinated bank holidays and coordinated delinking from major currencies. The idea of simulated delinking from major currencies raised some eyebrows because of its timing — on the same day participants gathered to launch “Collective Strength,” reports circulated that the Biden administration was considering removing Russia from the global electronic-payment-messaging system known as SWIFT, short for Society for Worldwide Interbank Financial Telecommunication.

This measure would be part of a package of economic sanctions the U.S. would levy against Russia should it attack Ukraine. However, what may raise even more eyebrows is the list of participants in the “Collective Strength” simulation, which includes: the IMF and World Bank, and indirectly, the World Economic Forum (WEF). It was the WEF, along with the Bill & Melinda Gates Foundation and the Johns Hopkins Bloomberg School of Public Health, which ran the simulated “Event 201” in October 2019. As previously reported by The Defender, the WEF also supported the development of financial instruments, such as credit and debit cards, that would track “personal carbon allowances” on an individualized basis.

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Michael Jackson

 

 

 

 

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