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Mr. House, I get the whole numerator/denominator, jigging relationships to look like hockey sticks instead of plateaus and yes they are lying to build back better or great reset. Or maybe the CCP virus plus uploaded CCP mRNA sequence is a set up for MERS-Cov 2.0. It’s just a thought experiment into unintended or intended consequences and how we may be blind to the outcome. As @Antidote rightly reminds us, “we only see what we look at”
Here is a presentation of a study that focuses on Medicare recipients 65 and over.
https://www.humetrix.com/powerpoint-vaccine.html
My thoughts on Chart 16
In the trials that established the baseline efficacy, people 65 and over weren’t included
Wasn’t there a lawyer that obtained death numbers in the Medicare system of people that died before they were considered fully jabbed up. How would that change this chart if we considered these as breakthroughs infections that were hospitalized?
And Chart 17
Impressive risk reduction for those who previously had Covid and got the jab. Likely, has more to do with natural immunity. This group of people are likely skewing the benefit on the previous chart.
But here is the thing, what if ADE is not a thing and nobody starts melting in their footprint like WTC7. In the limit, as more and more unvaccinated people that have had Covid get jabbed will the efficacy numbers start to inflect making boosters look like they are improving things. I know, Its really natural immunity that’s the driver but the data will be used to give credit to the jab.
Veracious Poet, 38 years ago I was 18 and on track to become a good corporate soldier. No wonder it feels like the Constitution and Bill of Rights are null and void. Your commentary is always appreciated even though the mood elevator can take a hit.
On our way out for a ride tonight, my wife was talking about not having carpets when we move. I said, “Babe, we might have to get used to dirt floors.”
But ya know, I feel a new kind of freedom. Made some new friends with some amazing people that under the old normal I would have never met. My wife’s mom lived with us her last year of life. If it wasn’t for Covid and the locking down of senior care facilities we would have never got that opportunity. Our ride ended with a double rainbow. How many of those do you see in a lifetime.
I am sure Louis experienced the some of the worst aspects of what his fellow man was capable of but yet it brought the best in him for all to enjoy.
We got our “get the jab email” on Friday.
In this email they said that we will follow the law. Sorry, this is not a law. It is an Executive Order from the President. It only covers federal employees under the direct control of the Executive Branch (DHS, DOD, DOJ, etc.). It only applies to those contractors that work directly for the federal government in the Executive Branch controlled agencies. It does NOT apply to privately owned defense contractor companies that have been awarded defense contracts. So our company lawyers are playing it a little loose with wording to force the mandate. That being said, if we ever want to win another US defense contract, the company will do what ever the government recommends, regardless of its ethical or legal standing.
They set the date for Dec. 8th. I don’t think that date is unique to us but was set by the Biden administration. Hey, one day after the Pearl Harbor day. They really want to liken this to a war.
Non-negotiable, never felt so alive.
@Raul “can you turn your research into an article? I can help with the writing if needed”
I would need your help, you have an amazing gift/talent for doing that.
TDK,
Will do, this infection can really set you back making the recovery frustrating. In article [1], the long term impact on WBCs was a new one for me. I haven’t dug too deep here yet. My wife is nearly recovered, still seems to have a bit of walking pneumonia and fatigue. Just got some blood work back and her thyroid was impacted. Big swing towards the hyper end of the range.
The human body is a universe in and of itself and each aspect of it is a galaxy.
In regards to IVM studies.
1) There may be little impact in a respiratory only infection, therefore taking it prophylactically may not help much in the respiratory phase if infected.
2) The real impact seems to be for those whose infections have moved into the vascular system.
Anyway, before addressing the best theory I have come across regarding Covid in the blood I wanted to circle back and highlight the spike proteins RBD affinity to Sialic Acid (SA) and its importance in the human body. Here is the image, showing how this affinity grows from a Corona cold virus thru Sars-Cov2.
Below is from the abstract of the linked paper. (It’s mainly for reference if interested)
[4] https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7153325/Sialic acid play important roles in human physiology of cell-cell interaction, communication, cell-cell signaling, carbohydrate-protein interactions, cellular aggregation, development processes, immune reactions, reproduction, and in neurobiology and human diseases in enabling the infection process by bacteria and virus, tumor growth and metastasis, microbiome biology, and pathology. It enables molecular mimicry in pathogens that allows them to escape host immune responses.
Viral sialic acid-recognizing lectins or HAs can agglutinate RBC. Viruses use sialic acids linked to glycoproteins and gangliosides to attach to host cells, followed by their entry, for example, corona virus, DNA tumor viruses, hepatitis virus, influenza viruses (A, B, and C), mouse polyoma virus, mumps, Newcastle disease virus (NDV), norovirus, parainfluenza viruses, rotavirus, and Sendai virus. HAs from influenza A, C, NDV, and polyoma viruses have been crystallized. Sialic acid-recognizing lectins from adenoviruses and picornaviruses have not been identified.
Some of these viruses carry neuraminidase or sialyl-O-acetyl-esterase that destroys the receptor, promotes virus release from infected cells, and removes sialic acid on host cell affecting cell surface binding of the virus. Influenza A virus enters the host by using host surface sialic acids. Influenza C virus HA-esterase specific for 9-O-acetylated sialic acids can break down 9-O-acetyl ester. HA-esterase from mouse hepatitis virus is specific to sialic acids substituted by O-acetyl group at the C-4 position (Neu4,5Ac2). HA-neuraminidase of NDV84 and parainfluenza viruses perform vital functions in infection biology
Back in Oct2020, independent researcher David Scheim, uploaded this paper to SSRN. It is not peer reviewed. In virus time it was before the rollout of the jabs and early days of mass IVM awareness. Here is the link to the abstract. From there you can open the pdf.
https://papers.ssrn.com/sol3/papers.cfm?abstract_id=3706347
Abstract;
The role of vascular occlusion in the morbidities, pulmonary and systemic, of COVID-19 has received increasing focus. Histological studies of lung tissue from COVID-19 patients have found extensively damaged endothelium of capillaries adjoining relatively intact alveoli, corresponding to hypoxemia accompanying normal breathing mechanics in such patients. Advanced image analysis of lung CT scans of COVID-19 patients reveals redistribution of blood flow from smaller to larger diameter blood vessels, this effect correlated with the degree of breathing dysfunction.
Essential to the study of vascular occlusion in COVID-19 are viral properties dating back to studies of Jonas Salk in the 1940s that have been positively established for SARS-CoV-2.
First, SARS-CoV-2 binds to red blood cells (RBCs), in vitro and also clinically in COVID-19 patients.
Second, although fusion and replication of SARS-CoV-2 occur via ACE2, such hemagglutinating viruses initially attach to infective targets and clump with blood cells via much more abundantly distributed sialic acid (SA) glycoconjugate binding sites. SARS-CoV-2, in particular, attaches to these SA sites.
Third, certain enveloped viruses express an enzyme, hemagglutinin esterase (HE), that counteracts viral-RBC clumping. Notably, among betacoronaviruses, the common cold strains express HE while SARS-CoV-2, SARS-CoV-1 and MERS, the virulent strains, do not.
These hemagglutinating properties of SARS-COV-2 establish a framework for “catch and clump” induction of microvascular occlusion proposed here. Ultramicroscopic studies of tissues from COVID-19 patients indicate a key role for hemagglutination early and mid-course in COVID-19, before such clumps harden into clots via the coagulation cascade. Hemagglutination may be reversed by two anti-COVID-19 therapeutics that each competitively bind to SARS-CoV-2 spike protein, blocking such viral attachments. One therapeutic is antiviral antibodies generated by vaccines, the anti-hemagglutination effect of which is exhibited in Jonas Salk’s hemagglutination inhibition assay. The other therapeutic is ivermectin (IVM), a drug of Nobel Prize honored distinction, distributed in 3.7 billion doses worldwide. In ten clinical trials, three with randomized controls, IVM yielded mortality reductions for COVID-19 of 90% at highest doses. IVM may limit virulence of SARS-CoV-2 by steric interference with multivalent spike protein attachments to SA binding sites, blocking hemagglutination, an effect likely to target mutant viral strains.
I had to switch from “be the spike” to “be the Lipid Nano Particle” and I now find myself in a vortex of understanding for which there may be no escape velocity. It could be a kool-aid vortex, but we will see. Suffice it to say, though the primary mechanisms are different, the understanding gained on the infection side is still most useful.
The inflection gains access to the blood stream (“ARDS” like DAD not a precursor)
1) The virus gains access to an epithelial cell in the air sac of the lungs by attaching to ACE2. It uses this cell to make many copies of itself.
2) Once it breaches the epithelial wall, which is only one cell thick it gains access to an ACE2 receptor on the pericytes, which sit on top of the endothelial cells that make up the nearby capillaries.
3) Many more copies are made, weaking the capillary walls, which again are only one cell thick. The virus now has access to the blood stream.Here are three interesting articles, starting with the most recent, describing complications related to having a spike coated virus in the blood.
[1] (virus time = Jul2021)
https://www.dicardiology.com/content/covid-19-changes-properties-blood-cells“Shortness of breath, fatigue and headaches, some patients still struggle with these long-term effects of a severe infection by the SARS-CoV-2 coronavirus after six months or more. This post COVID-19 syndrome, also called COVID long-haulers, is still not properly understood. What is clear is that during the course of the disease, often blood circulation is impaired, clotting and dangerous vascular occlusions can occur, and oxygen transport in is limited. These are all phenomena in which the blood cells and their physical properties play a key role.”
“They found that, for example, the size and deformability of the red blood cells of patients with the disease deviated strongly from those of healthy people. This indicates damage to these cells and could explain the increased risk of vascular occlusion and embolisms in the lungs. In addition, the oxygen supply, which is one of the main tasks of the erythrocytes, may be impaired in infected persons.”
Lymphocytes (one type of white blood cell responsible for the acquired immune defense) were in turn significantly softer in COVID-19 patients, which typically indicates a strong immune reaction. The researchers made similar observations for neutrophil granulocytes, another group of white blood cells involved in the innate immune response. These blood cells even remained drastically altered seven months after the acute infection.
[2] (virus time = Feb2021)
COVID-19 can affect the blood. Its spike protein may be the culprit.
In this article they refer to RGD, I think they mean RBD (Receptor Binding Domain)
The autopsy reports revealed COVID-19 patients were suffering from huge amounts of thick, coagulated blood, and dysfunctional blood vessels were tearing through body tissue instead of repairing it—highly uncommon side effects of respiratory diseases.
The postmortem evidence plus his own experience with something called “COVID toes”—an odd side effect of the disease that causes heightened blood vessel formation in the toes, turning them bright red—led Makowski to speculate that something about the virus might be causing abnormal blood-related complications.
“One of the most perplexing and devastating effects of this disease is the scenario where three or four weeks after being hospitalized with pneumonia, people under the age of 50 are back home, they feel fine, and then all of a sudden they have a stroke and die,” says Makowski, professor and chair of the bioengineering department at Northeastern.
[3] (virus time = Nov2020)
https://news.cuanschutz.edu/news-stories/attack-on-red-blood-cells-a-prime-suspect-in-covids-debilitating-effectsThe oxygen-saturation level of COVID patients, especially those with severe cases, was prone to dropping to dangerous levels, even below 90%. “We asked the question: Can it be due to the cell that transports oxygen?” D’Alessandro said. “Can COVID attack red blood cells – the most abundant cell in the human body – which has evolved specifically to transport oxygen?”
The answer to both questions was “yes.” The study revealed that SARS-CoV-2 damages the membranes of oxygen-carrying red blood cells. The virus didn’t affect the cells’ hemoglobin, which allow the cells to pick up oxygen, but it did damage membrane proteins responsible for blood cell structure, a characteristic that allows these cells to indirectly regulate red cell capacity to release oxygen and, most importantly, to squeeze through narrow capillaries in the periphery of the bloodstream.
“When the red blood cells are damaged, and you’re a COVID patient who is exposed to another stress – pharmacological treatment, high fever or, after recovering from the disease, exercise or something of that nature – then your red blood cell is more likely to hemolyze (rupture),” D’Alessandro said.
This explains why D’Alessandro, who jumped back into training for a late-summer marathon, noticed that more than a month after his initial COVID bout – two subsequent tests came out negative – he felt extremely tired just 20 minutes into a run.
“Something was happening in the red blood cells, and it’s why we believe that they are part of the problem in long-term COVID symptoms,” he said.The lingering oxygen-level difficulties are explained by the lifespan of red blood cells. The cells circulate for up to 120 days before the body replaces them. To make room for hemoglobin, red blood cells have evolved to lose nuclei and organelles that allow other cells to replace damaged molecular components. So, if the virus damages red blood cells, it will be up to four months before they are cleared and replaced with cells that do not carry such damage.
Dr. D = hot knife thru butter.l
A truly wise person uses few words; a person with understanding is even-tempered.
Question. In this Japanese government report on the bio distribution of lipid nano particles in rats, how should I interpret the blood related levels (bottom of chart) and someone’s decision not to report the % of Administered dose for the blood related levels (bottom right)?
Given that whole blood contains the liquid fraction of blood (i.e., plasma) as well as the cellular elements that lead to clotting under certain circumstances. Which include red blood cells (RBCs), white blood cells, and other components.
Should I smell a rat?
Kind of looking forward to the future. The last time oligarchical control was replaced following a pandemic with an emphasis on freedom of the human spirit was the Black Death.
Doc Robinson: “Intravenous injection of COVID-19 mRNA vaccine can induce acute myopericarditis in mouse model“, then what is a more likely (and better supported) cause of the damage?”
I think I will go for the “more likely” in the human model as we are meant to be the true guinea pigs. I have a few gaps in my theory. I either need to be shot down and put out of this quantum entanglement obsession I have with TAE or a bit of help filling in the final gaps.
Keep in mind my wheel house is aero/thermodynamics and nothing to do with this. I have been involved in more than my fair share of root/probable cause analyses so I am drawn to puzzles no matter what the subject matter. I think I have a grip on how to cast this. First I will state my theory and then back fill it with the supporting information as I get time to do so.
Basically this is the core of what I believe to be happening. Once the cells with surface spike glycoproteins enter the bloodstream, whether infection or injection, they will bind to the red and white blood cells. They will go thru the process of attaching and detaching depending on where they are in the vascular system. Clumps or clots can build up anywhere, precipitating any number of undesirable outcomes. The immune system does not particularly see them as invaders and they will eventually cycle out of our system as new cells are made to replace them. The measured immune response from the jab has little to do with preventing infection and also wanes as the life cycle of the impacted red and white blood cells is completed. The injections are worthless and they down regulate the immune system while the cells they produce are circulating.
Loved the responses from the NBA players to the press, especially Bradley Beal. I faintly heard the reporters amygdala short circuiting.
Biden to fine companies $70,000 To $700,000 for violations of Vaccine Mandate…
Posted by Kane on September 28, 2021 8:21 pmBiden’s Vax Mandate To Be Enforced By Fining Companies $70,000 To $700,000
On Saturday, Speaker Nancy Pelosi’s House quietly tucked an enforcement mechanism into their $3.5 trillion “reconciliation” bill, passed it out of the Budget Committee, and sent it to the House floor.
Buried on page 168 of the House Democrats’ 2,465-page mega bill is a tenfold increase in fines for employers that “willfully,” “repeatedly,” or even seriously violate a section of labor law that deals with hazards, death, or serious physical harm to their employees.
The increased fines on employers could run as high as $70,000 for serious infractions, and $700,000 for willful or repeated violations—almost three-quarters of a million dollars for each fine.
If enacted into law, vax enforcement could bankrupt non-compliant companies even more quickly than the $14,000 OSHA fine anticipated under Biden’s announced mandate.
My wife baited me into going to a freedom rally at our statehouse recently. She said there would be lots of nice looking nurses protesting the mandates. It was a beautiful day and flirting was easy. The protest was peaceful, though there was one guy there that had attached his American flag to a Louisville Slugger. Nice touch. I did get the opportunity to write a note to the governor and personally deliver it to his office.
I have been reading Walter Chestnut’s takes on the spike for a while. Don’t get me wrong, it is his ice and he can do triple axles around me all day long but it gets a little exasperating with all the sciencey dot connecting, whose to say if it he is right or wrong. Then again what isn’t the spike capable of? This last one though might be a nerdy attempt get a date with Nikki Manaj.
Did you see the Malone/Bossche interview with Dr. Phillips that germ posted recently? Bossche speaks and then Phillips looks to Malone to translate into caveman speak for the rest of us. The funny part was Malone has been letting his beard grow and is looking a bit like a caveman these days.
@Doc Robinson,
What if the spike is not pathogenic and we have simply projected our collective pathological fear and ignorance on it?What if the mRNA injections down regulate the immune system so that these surface glycoproteins aren’t seen as a threat. Actually, our immune system isn’t even alerted until there is enough of them and our defenses are down so they can mount an attack. Without the machinery for viral replication. Why would our immune system take issue with them or even care?
Another way to say it is that if I get the infection naturally my immune system is going to be like “hey I need to be on the look out for those glycoproteins on that sneaky devil”. But if I get the injection and there is really nothing pathogenic about the glycoproteins in and of themselves, then my immune my system will be lulled into a false sense of security when I encounter it or something like it in the future and a much higher viral load will be able to accumulate in my system. Isn’t this what we are seeing in the injected?
Meet the spike.
“The coronavirus sports a luxurious sugar coat. “It’s striking,” thought Rommie Amaro, staring at her computer simulation of one of the trademark spike proteins of SARS-CoV-2, which stick out from the virus’s surface. It was swathed in sugar molecules, known as glycans.
“When you see it with all the glycans, it’s almost unrecognizable,” says Amaro, a computational biophysical chemist at the University of California, San Diego.
Many viruses have glycans covering their outer proteins, camouflaging them from the human immune system like a wolf in sheep’s clothing.”
https://www.nature.com/articles/d41586-021-02039-y
So if we are only injecting the “sugar coat” and not the the whole enchilada, then how is that the immune system sees nothing more than a sheep?
In installment #4, I posted a TEDx video by professor Bertozzi where she discusses how these “sugar coated” cells communicate with our immune system. How they can literally put our immune cells to sleep. Today in the Project Veritas video that Raul posted today. There is a screen capture of the J&J scientist Justin (I have no moral compass) Durrant text that said the following.
“What I said about the formulations and cancer needs to stay between us. “
Anyways, I’m beginning to see a light at the end of the tunnel on how the spike leads to clotting in both the disease and the injection. But many times It’s not the right tunnel. We will see.
For those that have read this far, leave you with this throw back.
my parents,
They are creating a list of all who read and shared that one so they know who to round up. Just kidding, still not Australia.
Remember when the space shuttle burned up on reentry. It was due to damaged tiles that shield the vehicle from the extreme surface heating on reentry. The launch video captured pieces foam insulation hitting the shuttle. It was dismissed at first because it was foam. Eventually they built a cannon to fire a piece of foam of similar size and velocity. It damage the tiles.
In the moment, everyone has bought into the idea that the spike protein is pathogenic once it enters the blood stream. What if it isn’t?
@Doc Robinson,
Diameter of a typical (human) capillary is 0.001mm (Area=785,714 nm^2)
mRNA LNP diameter = 90-140nm (Average Area=10,391 nm^2)
Outer Diameter of a 25 gauge needle=0.515mm (500X the capillary diameter)
roughly 70 LNP can occupy the cross sectional area of a capillary.
Red Blood Cells move thru capillaries single file.
At any given moment, only about 5-10% of our capillary beds have blood flowing through them.
@Doc Robinson,
If injected IV, what type of cell(s) would be used would be used to build out a version with the spike protein?
650K reads / 1500 comments for Spartacus letter on ZH. Mostly affirmative comments and noise. A few contrarian ones of note.
“If SARS-CoV-2 is a virus similar to SARS it can not be a “blood disease”. A respiratory virus can not be a blood borne pathogen. It would also be nearly impossible to transmit through respiratory droplets. It is either a respiratory disease or it is not.”
“It is like “coronavirus” is the do-everything, pixie dust of the virus world. Like the symptoms range from the sniffles to your internal organs imploding……..but there’s “natural immunity”?”
“Funny how it “eats your blood vessels away” but the survival rate is 99.7%. Funny how it is a deadly bioweapon that is the greatest pandemic the world has ever seen, but if you are under 65 you have a better chance of dying from lightning strike.”
This entire “letter” is a thumbnail description of this psyop…….right down to its anonymity and choice of pseudonym.
Hmmm, has a Q-like vibe. Might be trying to punk us.
Spartacus 1st summary point:
“COVID-19 is a blood and blood vessel disease. SARS-CoV-2 infects the lining of human blood vessels, causing them to leak into the lungs.”
This where my understanding is currently.
On one side, we have the virus with all replicating machinery covered with surface spike glycoproteins that have a receptor binding domain that can attach to a cell expressing the ACE2.
On the other side, we have injection that can create a non replicating version of this virus with spike glycoproteins on the surface which in theory can mimic the receptor binding domain properties of the spike glycoprotein on the virus.
Endothelial make up our vascular plumbing and can express this ACE2 receptor. This could be bad if the replicating virus where to attach to this receptor.
What does it mean if the non replicating version were to make it into the vascular system? With no replicating machinery, how does it damage the endothelial cell? Seems like a nothing burger in this case.
In a live person (not so close to death) blood travels in the heart around 25 cm/s (10 in/s) on average. The diameter of the virus is on the order of 100nm. In my math is right, then virus would be moving 2.5 million body lengths every second. In the moment, I have a hard time believing that the small patch in the spike receptor binding domain has any chance of attaching to an ACE2 receptor in the heart.
Here are the similarities between the respiratory infection and the mRNA injection. The infection attacks the epithelial cells in the air sacs ultimately gaining access to the nearby capillaries.
In the intramuscular injection, non replicating versions of the virus are created and somehow gain access to nearby capillaries.
I am working on putting a floor under my working theory. The links that Raul and Germ have posted over the last few days have helped fill in some gaps that I was wrestling with. And this cancer stuff, holy heck. It’s been fascinating path of discovery overall, even if I come up bagel.
Raul, thanks again for the line up today.
In the limit, as we provide opportunities for the virus to optimize it’s symbiotic relationship with our immune system we are likely to live long miserable immunocompromised lives, irrespective of injection status. Until, of course, some of the sickly survivors mate up produce a gene in their offspring that provides protection.
Spikes are Bad and Spikes in the Blood are Really Bad (installment 4 of ??)
Here is my current working theory.
The virus will attach to a host cell that has an ACE2 receptor using is surface spike protein. Once inside the host cell, hidden from the immune system, it will make many copies of itself. The host cell dies and now there are many new copies of the virus available to attach to more host cells. Cells with ACE2 receptors are found throughout the body.
The spike protein is also capable of binding to Sialic Acid (SA). The blood is a target rich environment for SA. Red blood cells (RBCs) are coated with it. Once attached to an RBC, it can move throughout the body until conditions are favorable for it to attach to a call expressing ACE2. The affinity for the spike to bind to ACE2 is much greater than SA. Fluidic forces, like turbulence, could free the virus from the RBC, possibly damaging it. Also in regions of slow blood flow and high virus count, RBCs and the viruses could clump up due to the SA binding properties. This would impede blood flowing in that region ultimately causing vascular damage.
Though this video is about cancer, there is enlightening info about SA.
I first started to consider the role of SA in a Covid infection after seeing the figure below. These are some of the viruses that are in the corona virus family. It shows the different types of host receptors for binding. Also, shown is the growing affinity for SA going from left to right.
The corresponding paper is linked here.
Thanks for response yesterday Funny we caught Covid shortly after setting up camp along Lake Michigan. The beach/bike vacations are our favorites.
I split time between a old Trek MT820 and a Trek Navigator 3.0. I estimate I have put about 15k miles on the navigator, a real workhorse. For MTB, I have a hard tail Rockhopper. Haven’t road biked much. Picked up an old Technium Tri-lite. The story behind the Tri-lite is that Boeing and Raleigh engineers were having lunch. The Boeing guys let on to the fact that they had a good process for thermally bonding steel and aluminum thus eliminating the weld. So the steel lugs/BB are thermally bonded to the aluminum top, bottom and seat tubes on this one.
Anyway, love the bike, riding, fixing or reconditioning in my spare time. Picked up a couple dozen to refurb over the winter. Hope I get the time.
Spikes are Bad and Spikes in the Blood are Really Bad (installment 3 of ??)
Installment #1 summary: There are certain factors, on the surface of cells, that mediate the entry of the spike protein. These have been identified as ACE2, TMPRSS2, Salic Acid (SA), CD147, cathespin B and L. All these factors involved in the entry of the spike protein have been shown to be expressed by the endothelial cells. Endothelial cells make up our vascular plumbing. They also make up our lymphatic plumbing.
https://encyclopedia.pub/1397Installment #2 summary: Animation of influenza virus invading the lungs
The clot thickens.
“Autopsies on people who died of the coronavirus are helping doctors understand how the disease affects the body — and one of the most remarkable findings concerned blood clotting, a pathologist says. Dr. Amy Rapkiewicz, the chairman of the department of pathology at NYU Langone Medical Center, spoke to Erin Burnett on OutFront (CNN) Thursday night (7/9/2020).
Some Covid-19 patients are known to develop blood clotting issues, but the degree and the extent to which that occurs was described as “dramatic” by Rapkiewicz.
In the early stages of the pandemic, bedside clinicians noticed a lot of blood clotting “in lines and various large vessels,” she said. “What we saw at autopsy was sort of an extension of that,” she said. “The clotting was not only in the large vessels but also in the smaller vessels.
“And this was dramatic, because though we might have expected it in the lungs, we found it in almost every organ that we looked at in our autopsy study,” she said. Rapkiewicz’s study outlining her findings was published at the end of June (2020) in The Lancet journal EClinicalMedicine.”
So, we are about 6 months into this thing, and they are finding that in severe cases the damage extends well beyond the lungs.Fun fact: According to Steve Black (M.B.Ch.B D.A. FFARC/P Medical Practitioner & Anaesthesiologist, Pilot, University of Leeds) The arm brain circulation time, which is the time taken for an intravenous injection [not intramuscular] in the arm [assume wrist] to pass through the heart and lungs and out up to the brain through the carotid arteries, is around 15 to 20 seconds.
Diffuse alveolar damage (DAD) is characteristically seen in severe cases. Here is a schematic an alveolar sac.
Notice how the epithelium (air sac wall) and the endothelium (capillary wall) are simply one cell thick.
Here is a video that describes the how this virus reeks havoc, not only on the air sac walls but also the capillary walls rendering them leaky. This would provide a pathway for the virus (spike) to enter the blood stream. Keep in mind this video made was early days (Mar2020) prior to Dr. Amy Rapkiewicz comments above regarding the autopsy evidence of clotting throughout the body.@Boogaloo I just reordered. We burned thru most of what we had with our recent bout with Covid. A friend of my son’s had dropped off fire pit and was wondering how orally dose some injectable that he had picked up.
Any comments on taking injectable ivermectin orally? Jokes are welcome too. It’s a 1% sterile solution for cattle. Cattle dosage is 1ml per 110# of body weight. I have a friend that is scrambling to find something before the next wave and it was the only product available at a local tractor supply.
Spikes are Bad and Spikes in the Blood are Really Bad (installment 2 of ??)
Installment #1 summary: There are certain factors, on the surface of cells, that mediate the entry of the spike protein. These have been identified as ACE2, TMPRSS2, Salic Acid (SA), CD147, cathespin B and L. All these factors involved in the entry of the spike protein have been shown to be expressed by the endothelial cells. Endothelial cells make up our vascular plumbing. They also make up our lymphatic plumbing.
In general, viral invaders have surface proteins that facilitate entry into the host cell. Influenza has two, HA and NA. Here is a nice animation that demonstrates how these invaders attack the lungs.
Spikes are Bad and Spikes in the Blood are Really Bad (installmentsd 1 of ??)
I am more accustomed to the language of aero-thermo fluid dynamics and not so much biology. Basically, I am no expert and just want to unpack the why’s. It would be nice to have a Bob Malone to bounce things off of, but I think this community that Raul has created may actually be best.
Anyways, understanding the spikes are the key whether natural or injected. The following image gives a good visual of what kind of receptors the spikes have an affinity for. More detail can be found in the link to this paper.
Thanks Doc Robinson,
So In the case of the mRNA injection is it the instructions that make a spike or does the manufactured spike remain encapsulated?
“Treat Your Own Covid” blog probably saved a view lives
Who do I thank?
Give me Covid naturally or give death.
I mean liberty
#12 in the should get vaccinated list
“Defective virus design (s1 was never supposed to be free, inclusion of PEG was unnecessary and allows LNP to be widely distributed)“
Does anybody have good links to info that would shore up this statement? I’m coming up bagel in my search. Thanks
If you look forward to and enjoy Dr. D’s commentary everyday, this linked article/interview has that vibe.
https://www.gatestoneinstitute.org/17695/hinge-moment-history
